Developmental nicotine exposure results in programming of alveolar simplification and interstitial pulmonary fibrosis in adult male rats

Reproductive Toxicology

Volumn 34, Issue 3, 2012, Pages 370-377

  Dasgupta, Chiranjib ^a,b   Xiao, DaLiao ^a   Xu, Zhice ^a,b   Yang, Shumei ^c   Zhang, Lubo ^a,b  

^a THE FIRST AFFILIATED HOSPITAL OF SOOCHOW UNIVERSITY   (China)

^b LOMA LINDA UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c CALIFORNIA STATE UNIVERSITY   (United States)

Author keywords

Angiotensin receptor; Lung; Nicotine; Programming

Indexed keywords

ANGIOTENSIN 1 RECEPTOR; ANGIOTENSIN 2 RECEPTOR; COLLAGEN; CONNECTIVE TISSUE GROWTH FACTOR; NICOTINE; SMAD3 PROTEIN; TRANSFORMING GROWTH FACTOR BETA1;

ALVEOLAR SEPTAL THICKNESS; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BODY WEIGHT; CONTROLLED STUDY; FEMALE; FIBROSING ALVEOLITIS; LUNG ALVEOLUS; LUNG BODY WEIGHT RATIO; LUNG DEVELOPMENT; LUNG VOLUME; LUNG WEIGHT; MALE; NONHUMAN; PREGNANCY; PRENATAL EXPOSURE; PROGENY; PROTEIN EXPRESSION; RADIAL ALVEOLAR COUNT; RAT; RESPIRATORY TRACT PARAMETERS;

ANIMALS; COLLAGEN; FEMALE; LUNG DISEASES, INTERSTITIAL; MALE; MATERNAL-FETAL EXCHANGE; NICOTINE; NICOTINIC AGONISTS; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; PULMONARY FIBROSIS; RATS; RATS, SPRAGUE-DAWLEY; RECEPTOR, ANGIOTENSIN, TYPE 1; RECEPTOR, ANGIOTENSIN, TYPE 2; TRANSFORMING GROWTH FACTOR BETA1;

ANIMALIA; RATTUS;

EID: 84865544075     PISSN: 08906238     EISSN: 18731708     Source Type: Journal    
DOI: 10.1016/j.reprotox.2012.05.100     Document Type: Article

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