![]() |
Volumn 17, Issue 2, 2012, Pages 311-320
|
Reactive-oxygen-species-mediated Cdc25C degradation results in differential antiproliferative activities of vanadate, tungstate, and molybdate in the PC-3 human prostate cancer cell line
|
Author keywords
Cell cycle arrest; Molybdate; Reactive oxygen species; Tungstate; Vanadate
|
Indexed keywords
CDC25A PROTEIN;
CDC25B PROTEIN;
CDC25C PROTEIN;
CELL CYCLE PROTEIN;
CYCLIN DEPENDENT KINASE 1;
MOLYBDIC ACID;
REACTIVE OXYGEN METABOLITE;
TUNGSTEN DERIVATIVE;
TUNGSTIC ACID;
UNCLASSIFIED DRUG;
VANADIC ACID;
ANTINEOPLASTIC AGENT;
MOLYBDENUM;
PROTEIN TYROSINE PHOSPHATASE;
ANTIPROLIFERATIVE ACTIVITY;
ARTICLE;
CANCER CELL;
CELL CYCLE ARREST;
CELL CYCLE G2 PHASE;
CELL CYCLE M PHASE;
CONTROLLED STUDY;
GROWTH INHIBITION;
HUMAN;
HUMAN CELL;
PRIORITY JOURNAL;
PROSTATE CANCER;
PROTEIN DEGRADATION;
PROTEIN PHOSPHORYLATION;
CELL CYCLE CHECKPOINT;
CELL PROLIFERATION;
DRUG ANTAGONISM;
DRUG EFFECT;
MALE;
METABOLISM;
PROSTATE TUMOR;
TUMOR CELL LINE;
ANTINEOPLASTIC AGENTS;
CDC25 PHOSPHATASES;
CELL CYCLE CHECKPOINTS;
CELL LINE, TUMOR;
CELL PROLIFERATION;
HUMANS;
MALE;
MOLYBDENUM;
PROSTATIC NEOPLASMS;
REACTIVE OXYGEN SPECIES;
TUNGSTEN COMPOUNDS;
VANADATES;
|
EID: 84861213221
PISSN: 09498257
EISSN: 14321327
Source Type: Journal
DOI: 10.1007/s00775-011-0852-1 Document Type: Article |
Times cited : (41)
|
References (43)
|