The genetic basis of primary aldosteronism

Current Hypertension Reports

Volumn 14, Issue 2, 2012, Pages 120-124

  Funder, John W ^a  

^a HUDSON INSTITUTE OF MEDICAL RESEARCH   (Australia)

Author keywords

Adrenal adenoma; Adrenal zonation; Bilateral adrenal hyperplasia; Familial hyperaldosteronism; FH 1; FH II; FH III; Genetics; Germline mutation; Hypertension; KCNJ5; Primary aldosteronism; Somatic mutation; TASK 1; TASK 3; Zona fasciculata; Zona glomerulosa

Indexed keywords

ALDOSTERONE; BETA CATENIN; CRYPTOCHROME 1; CRYPTOCHROME 2; POTASSIUM CHANNEL; TWIK RELATED ACID SENSITIVE POTASSIUM CONTAINING CHANNEL 1; TWIK RELATED ACID SENSITIVE POTASSIUM CONTAINING CHANNEL 3; UNCLASSIFIED DRUG;

ADRENAL CORTEX; ALDOSTERONE RELEASE; ARTICLE; CLINICAL FEATURE; DIAGNOSTIC PROCEDURE; DISEASE MODEL; FAMILIAL HYPERALDOSTERONISM TYPE 1; FAMILIAL HYPERALDOSTERONISM TYPE 2; FAMILIAL HYPERALDOSTERONISM TYPE 3; GENE; GENE EXPRESSION; GENETIC ANALYSIS; HUMAN; KCNJ5 GENE; MOLECULAR MECHANICS; NONHUMAN; POLYMERASE CHAIN REACTION; PREVALENCE; PRIMARY HYPERALDOSTERONISM; SOMATIC MUTATION;

G PROTEIN-COUPLED INWARDLY-RECTIFYING POTASSIUM CHANNELS; GENETIC PREDISPOSITION TO DISEASE; GERM-LINE MUTATION; HUMANS; HYPERALDOSTERONISM; NERVE TISSUE PROTEINS; POTASSIUM CHANNELS, TANDEM PORE DOMAIN; RISK FACTORS;

EID: 84861102187     PISSN: 15226417     EISSN: 15343111     Source Type: Journal    
DOI: 10.1007/s11906-012-0255-x     Document Type: Article

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