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Volumn 331, Issue 6018, 2011, Pages 685-686
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The genetics of primary aldosteronism
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Author keywords
[No Author keywords available]
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Indexed keywords
ALDOSTERONE;
ANGIOTENSIN 1 RECEPTOR;
GLUCOCORTICOID;
INWARDLY RECTIFYING POTASSIUM CHANNEL;
POTASSIUM CHANNEL;
G PROTEIN COUPLED INWARDLY RECTIFYING POTASSIUM CHANNEL;
KCNJ5 PROTEIN, HUMAN;
GENOMICS;
MUTATION;
POTASSIUM;
STEROID;
TUMOR;
ADRENAL TUMOR;
CHROMOSOME 7;
CIRCADIAN RHYTHM;
ELECTROPHYSIOLOGY;
ESSENTIAL HYPERTENSION;
GENE DELETION;
GENE MUTATION;
GENETIC ANALYSIS;
GENETICS;
HUMAN;
NONHUMAN;
PHENOTYPE;
POTASSIUM EXCRETION;
PRIMARY HYPERALDOSTERONISM;
PRIORITY JOURNAL;
SHORT SURVEY;
SODIUM RETENTION;
TUMOR VOLUME;
ADRENAL CORTEX ADENOMA;
ADRENAL CORTEX TUMOR;
ADRENAL GLAND;
ANIMAL;
DISEASE MODEL;
FEMALE;
HYPERALDOSTERONISM;
HYPERPLASIA;
HYPERTENSION;
MALE;
METABOLISM;
MOUSE;
MUTATION;
NOTE;
PATHOLOGY;
PATHOPHYSIOLOGY;
ADRENAL CORTEX NEOPLASMS;
ADRENAL GLANDS;
ADRENOCORTICAL ADENOMA;
ALDOSTERONE;
ANIMALS;
DISEASE MODELS, ANIMAL;
FEMALE;
G PROTEIN-COUPLED INWARDLY-RECTIFYING POTASSIUM CHANNELS;
HUMANS;
HYPERALDOSTERONISM;
HYPERPLASIA;
HYPERTENSION;
MALE;
MICE;
MUTATION;
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EID: 79951501330
PISSN: 00368075
EISSN: 10959203
Source Type: Journal
DOI: 10.1126/science.1202887 Document Type: Short Survey |
Times cited : (28)
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References (12)
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