GFAP-Cre-mediated transgenic activation of Bmi1 results in pituitary tumors

PLoS ONE

Volumn 7, Issue 5, 2012, Pages

  Westerman, Bart A ^a,b   Blom, Marleen ^a   Tanger, Ellen ^a   van der Valk, Martin ^a   Song, Ji Ying ^a   van Santen, Marije ^b   Gadiot, Jules ^a   Cornelissen Steijger, Paulien ^a   Zevenhoven, John ^a   Prosser, Haydn M ^c   Uren, Anthony ^a,d   Aronica, Eleonora ^b   van Lohuizen, Maarten ^a,e  

^a NETHERLANDS CANCER INSTITUTE   (Netherlands)

^b ACADEMIC MEDICAL CENTER   (Netherlands)

^c WELLCOME TRUST SANGER INSTITUTE   (United Kingdom)

^d MRC CLINICAL SCIENCES CENTRE   (United Kingdom)

^e NETHERLANDS CANCER INSTITUTE   (Netherlands)

Author keywords

[No Author keywords available]

Indexed keywords

BETA ENDORPHIN; BMI1 PROTEIN; CORTICOTROPIN; CRE RECOMBINASE; CYCLIN DEPENDENT KINASE INHIBITOR 2A; GLIAL FIBRILLARY ACIDIC PROTEIN; PROTEIN P19; PROTEIN P53; SYNAPTOPHYSIN; BMI1 PROTEIN, MOUSE; INTEGRASE; NUCLEAR PROTEIN; ONCOPROTEIN; REPRESSOR PROTEIN; RETINOBLASTOMA PROTEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CANCER SUSCEPTIBILITY; CELL LINEAGE; CONTROLLED STUDY; GENE ACTIVATION; GENE OVEREXPRESSION; HYPOPHYSIS TUMOR; IMMUNOHISTOCHEMISTRY; MEDULLOBLASTOMA; MOUSE; NONHUMAN; PROTEIN ANALYSIS; PROTEIN DEPLETION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; TRANSGENIC MOUSE; ANIMAL; GENE EXPRESSION; GENETICS; HUMAN; METABOLISM; PATHOLOGY; TRANSGENE;

MUS;

ANIMALS; BETA-ENDORPHIN; CYCLIN-DEPENDENT KINASE INHIBITOR P16; GENE EXPRESSION; GLIAL FIBRILLARY ACIDIC PROTEIN; HUMANS; INTEGRASES; MEDULLOBLASTOMA; MICE; MICE, TRANSGENIC; NUCLEAR PROTEINS; PITUITARY NEOPLASMS; PROTO-ONCOGENE PROTEINS; REPRESSOR PROTEINS; RETINOBLASTOMA PROTEIN; TRANSGENES; TUMOR SUPPRESSOR PROTEIN P53;

EID: 84860535452     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0035943     Document Type: Article

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