Delayed calcium dysregulation in neurons requires both the NMDA receptor and the reverse Na+/Ca2+ exchanger

Neurobiology of Disease

Volumn 46, Issue 1, 2012, Pages 109-117

  Brittain, Matthew K ^a   Brustovetsky, Tatiana ^a   Sheets, Patrick L ^a,b   Brittain, Joel M ^a   Khanna, Rajesh ^a   Cummins, Theodore R ^a   Brustovetsky, Nickolay ^a  

^a INDIANA UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b NORTHWESTERN UNIVERSITY   (United States)

Author keywords

Calcium dysregulation; Excitotoxicity; Glutamate; Na+ Ca2+ exchanger; Neuron; NMDA receptor

Indexed keywords

N METHYL DEXTRO ASPARTIC ACID RECEPTOR; SODIUM CALCIUM EXCHANGE PROTEIN;

ANIMAL CELL; ARTICLE; BRAIN NERVE CELL; CELL MEMBRANE DEPOLARIZATION; CELL MEMBRANE POTENTIAL; CELLULAR PARAMETERS; CONTROLLED STUDY; DELAYED CALCIUM DYSREGULATION; EXCITOTOXICITY; ION CURRENT; NERVE CELL NECROSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; RAT; SODIUM CALCIUM EXCHANGE; SODIUM CELL LEVEL; TRANSPORT KINETICS;

ANIMALS; CALCIUM; GLUTAMIC ACID; HIPPOCAMPUS; NEURONS; NEUROPROTECTIVE AGENTS; PRIMARY CELL CULTURE; RATS; RECEPTORS, N-METHYL-D-ASPARTATE; SODIUM-CALCIUM EXCHANGER;

EID: 84858161293     PISSN: 09699961     EISSN: 1095953X     Source Type: Journal    
DOI: 10.1016/j.nbd.2011.12.051     Document Type: Article

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