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Volumn 18, Issue 10, 2011, Pages 1545-1546
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Can the analysis of BH3-only protein knockout mice clarify the issue of direct versus indirect activation of Bax and Bak
a
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Author keywords
[No Author keywords available]
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Indexed keywords
BH3 PROTEIN;
BIM PROTEIN;
GLUCOCORTICOID;
PROTEIN BAK;
PROTEIN BAX;
PROTEIN BCL 2;
PROTEIN BID;
PUMA PROTEIN;
APOPTOSIS REGULATORY PROTEIN;
CYTOCHROME C;
MEMBRANE PROTEIN;
TUMOR SUPPRESSOR PROTEIN;
APOPTOSIS;
CELL DEATH;
CELL SURVIVAL;
CYTOTOXICITY;
DNA DAMAGE;
EDITORIAL;
ENZYME ACTIVATION;
EXPERIMENTAL MOUSE;
HEMATOPOIETIC CELL;
IN VITRO STUDY;
IN VIVO STUDY;
KNOCKOUT MOUSE;
LYMPHOCYTE;
NEUROLOGIC DISEASE;
NONHUMAN;
PHENOTYPE;
PRIORITY JOURNAL;
PROTEIN BINDING;
PROTEIN PHOSPHORYLATION;
STIMULUS;
UPREGULATION;
VAGINA;
ANIMAL;
CHEMISTRY;
DEFICIENCY;
GENETICS;
INTRACELLULAR MEMBRANE;
METABOLISM;
MITOCHONDRION;
MOUSE;
PROTEIN MULTIMERIZATION;
MUS;
ANIMALS;
APOPTOSIS REGULATORY PROTEINS;
BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN;
BCL-2-ASSOCIATED X PROTEIN;
BH3 INTERACTING DOMAIN DEATH AGONIST PROTEIN;
CYTOCHROMES C;
INTRACELLULAR MEMBRANES;
MEMBRANE PROTEINS;
MICE;
MICE, KNOCKOUT;
MITOCHONDRIA;
PROTEIN MULTIMERIZATION;
TUMOR SUPPRESSOR PROTEINS;
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EID: 80052773825
PISSN: 13509047
EISSN: 14765403
Source Type: Journal
DOI: 10.1038/cdd.2011.100 Document Type: Editorial |
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Times cited : (31)
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References (12)
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