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Journal of Biological Inorganic Chemistry

Volumn 16, Issue 5, 2011, Pages 809-816

The mechanism for heme to prevent Aβ1-40aggregation and its cytotoxicity

(8) Bao, Qingui a Luo, Ying a Li, Wei a Sun, Xiaobo b Zhu, Cuiqing b Li, Pingwei c Huang, Zhong Xian a Tan, Xiangshi a

a FUDAN UNIVERSITY (China)

b FUDAN UNIVERSITY (China)

c TEXAS A AND M UNIVERSITY (United States)

Author keywords

; Alzheimer's disease; Amyloid aggregation; Cytotoxicity; Heme; Keywords Ab1 40

Indexed keywords

AMYLOID BETA PROTEIN[1-40]; COPPER ION; HEME; HYDROGEN PEROXIDE; PEROXIDASE; REACTIVE OXYGEN METABOLITE;

ALPHA HELIX; ANIMAL CELL; ARTICLE; BRAIN NERVE CELL; CATALYSIS; CELL VIABILITY; CIRCULAR DICHROISM; COMPLEX FORMATION; CONTROLLED STUDY; CYTOTOXICITY; DIFFERENTIAL PULSE VOLTAMMETRY; EMBRYO; ENZYME ACTIVITY; HIPPOCAMPUS; NONHUMAN; PRIORITY JOURNAL; PROTEIN AGGREGATION; PROTEIN PROTEIN INTERACTION; RAT; ULTRAVIOLET SPECTROSCOPY;

ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; CELL LINE; CELL SURVIVAL; CELLS, CULTURED; COPPER; HEME; HIPPOCAMPUS; HUMANS; NEURONS; OXIDATIVE STRESS; PEPTIDE FRAGMENTS; PEROXIDASE; RATS; RECOMBINANT PROTEINS;

RATTUS;

EID: 80051549718 PISSN: 09498257 EISSN: 14321327 Source Type: Journal
DOI: 10.1007/s00775-011-0783-x Document Type: Article

Times cited : (27)

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