A substance P antagonist improves outcome when administered 4 h after onset of ischaemic stroke

Brain Research

Volumn 1393, Issue , 2011, Pages 84-90

  Turner, Renée J ^a,b   Helps, Stephen C ^a,b   Thornton, Emma ^a,b   Vink, Robert ^a,b  

^a UNIVERSITY OF ADELAIDE   (Australia)

^b HANSON INSTITUTE   (Australia)

Author keywords

Ischaemic stroke; Neurogenic inflammation; Neuropeptides; NK1 tachykinin receptor antagonist; Oedema

Indexed keywords

N ACETYLTRYPTOPHAN; SODIUM CHLORIDE; SUBSTANCE P;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLOOD BRAIN BARRIER; BRAIN EDEMA; BRAIN INFARCTION SIZE; BRAIN ISCHEMIA; CONTROLLED STUDY; DRUG EFFECT; IMMUNOREACTIVITY; MALE; MEMBRANE PERMEABILITY; MIDDLE CEREBRAL ARTERY OCCLUSION; NONHUMAN; PRIORITY JOURNAL; RAT; REPERFUSION; TREATMENT OUTCOME;

ANIMALS; BLOOD-BRAIN BARRIER; BRAIN EDEMA; BRAIN ISCHEMIA; DISEASE MODELS, ANIMAL; DRUG ADMINISTRATION SCHEDULE; INFARCTION, MIDDLE CEREBRAL ARTERY; MALE; PROTEASE INHIBITORS; RATS; RATS, SPRAGUE-DAWLEY; RECEPTORS, NEUROKININ-1; RECOVERY OF FUNCTION; SUBSTANCE P; TIME FACTORS; TRYPTOPHAN;

ANIMALIA; RATTUS;

EID: 79955939854     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.brainres.2011.03.066     Document Type: Article

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