A ligand-receptor pair that triggers a non-apoptotic form of programmed cell death

Cell Death and Differentiation

Volumn 9, Issue 8, 2002, Pages 807-817

  Castro Obregon S ^a   del Rio, G ^a   Chen, S F ^a   Swanson, R A ^b   Frankowski, H ^a   Rao, R V ^a   Stoka, V ^c   Vesce, S ^a   Nicholls, D G ^a   Bredesen, D E ^a  

^a BUCK INSTITUTE FOR RESEARCH ON AGING   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c JO EF STEFAN INSTITUTE   (Slovenia)

Author keywords

Caspase independent cell death; Neurokinin 1 receptor; Neuronal death; Non apoptotic programmed cell death; Substance P

Indexed keywords

3 [3,5 BIS(TRIFLUOROMETHYL)BENZYLOXY] 2 PHENYLPIPERIDINE; 3,5 BIS(TRIFLUOROMETHYL)BENZYL N ACETYLTRYPTOPHAN; 3,5-BIS(TRIFLUOROMETHYL)BENZYL N-ACETYLTRYPTOPHAN; CASPASE; DRUG DERIVATIVE; ENZYME INHIBITOR; LIPOCORTIN 5; NEUROKININ 1 RECEPTOR; PIPERIDINE DERIVATIVE; PROTEIN SYNTHESIS INHIBITOR; SUBSTANCE P; TRYPTOPHAN; FAS ANTIGEN; LIGAND; NEUROPEPTIDE; NEUROPEPTIDE RECEPTOR; SUBSTANCE P RECEPTOR; TUMOR NECROSIS FACTOR RECEPTOR 1;

ANIMAL; APOPTOSIS; ARTICLE; CELL CULTURE; CELL SIZE; DRUG ANTAGONISM; DRUG EFFECT; ELECTRON MICROSCOPY; EPITHELIUM CELL; FETUS; FOREBRAIN; GENETICS; HUMAN; IMMUNOHISTOCHEMISTRY; KIDNEY; METABOLISM; NERVE CELL; PHYSIOLOGY; RAT; SPRAGUE DAWLEY RAT; ULTRASTRUCTURE; ANIMAL CELL; BRAIN CELL; CENTRAL NERVOUS SYSTEM; CONTROLLED STUDY; DEPRESSION; EMBRYO; ENZYME ACTIVATION; GENE EXPRESSION; HIPPOCAMPUS; HUMAN CELL; NONHUMAN; PAIN; PERIPHERAL NERVOUS SYSTEM; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; STRIATE CORTEX;

ANIMALS; ANNEXIN A5; APOPTOSIS; CASPASES; CELL SIZE; CELLS, CULTURED; ENZYME INHIBITORS; EPITHELIAL CELLS; FETUS; HUMANS; IMMUNOHISTOCHEMISTRY; KIDNEY; MICROSCOPY, ELECTRON; NEURONS; PIPERIDINES; PROSENCEPHALON; PROTEIN SYNTHESIS INHIBITORS; RATS; RATS, SPRAGUE-DAWLEY; RECEPTORS, NEUROKININ-1; SUBSTANCE P; TRYPTOPHAN; ANIMAL; ANNEXIN V; HUMAN; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0036024068     PISSN: 13509047     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.cdd.4401035     Document Type: Article

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