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Volumn 286, Issue 18, 2011, Pages 16402-16413

Phosphomimetic substitution of heterogeneous nuclear ribonucleoprotein A1 at serine 199 abolishes AKT-dependent internal ribosome entry site-transacting factor (ITAF) function via effects on strand annealing and results in mammalian target of rapamycin complex 1 (mtORC1) inhibitor sensitivity

Author keywords

[No Author keywords available]

Indexed keywords

AKT PHOSPHORYLATION; CELLULAR RESPONSE; COMPLEX 1; CYCLIND1; G1 ARREST; GLIOBLASTOMAS; HETEROGENEOUS NUCLEAR RIBONUCLEOPROTEIN (HNRNP); IN-CELL; IN-VITRO; INTERNAL RIBOSOME ENTRY SITE; MAMMALIAN TARGET; MAMMALIAN TARGET OF RAPAMYCIN (MTOR); NUCLEAR RIBONUCLEOPROTEIN; OVER-EXPRESSION; RAPAMYCIN; RELATIVE ACTIVITIES; REPORTER ASSAY; RNA-BINDING PROTEIN; SALVAGE PATHWAY; STRAND ANNEALING; TUMOR CELLS; VIA EFFECT;

EID: 79955537824     PISSN: 00219258     EISSN: 1083351X     Source Type: Journal    
DOI: 10.1074/jbc.M110.205096     Document Type: Article
Times cited : (24)

References (39)


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.