The proapoptotic member of the Bcl-2 family Bcl-2/E1B-19K-interacting protein 3 is a mediator of caspase-independent neuronal death in excitotoxicity

FEBS Journal

Volumn 278, Issue 1, 2011, Pages 134-142

  Zhang, Zhengfeng ^a,b   Shi, Ruoyang ^a   Weng, Jiequn ^a   Xu, Xingshun ^c   Li, Xin Min ^a   Gao, Tian Ming ^d   Kong, Jiming ^a,d  

^a UNIVERSITY OF MANITOBA   (Canada)

^b CHONGQING MEDICAL UNIVERSITY   (China)

^c SOOCHOW UNIVERSITY   (China)

^d SOUTHERN MEDICAL UNIVERSITY   (China)

Author keywords

apoptosis; Bcl 2 E1B 19K interacting protein 3 (BNIP3); caspase independent cell death; excitotoxicity; neuron

Indexed keywords

CASPASE; GLUTAMIC ACID; PROTEIN BNIP3;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL VIABILITY; CONTROLLED STUDY; EMBRYO; ENZYME ACTIVITY; EXCITOTOXICITY; HIPPOCAMPUS; IN VITRO STUDY; IN VIVO STUDY; MALE; NERVE CELL NECROSIS; NEUROMODULATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; PROTEIN SYNTHESIS; PROTEIN TARGETING; RAT; RNA INTERFERENCE; UPREGULATION;

ANIMALS; APOPTOSIS; BLOTTING, WESTERN; CASPASES; CELLS, CULTURED; GENE EXPRESSION REGULATION; IMMUNOHISTOCHEMISTRY; MALE; MEMBRANE PROTEINS; NEURONS; PROTO-ONCOGENE PROTEINS; RATS; RATS, SPRAGUE-DAWLEY; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;

EID: 78650449393     PISSN: 1742464X     EISSN: 17424658     Source Type: Journal    
DOI: 10.1111/j.1742-4658.2010.07939.x     Document Type: Article

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