Aryl hydrocarbon receptor nuclear translocator in hepatocytes is required for aryl hydrocarbon receptor-mediated adaptive and toxic responses in liver

Toxicological Sciences

Volumn 118, Issue 2, 2010, Pages 554-563

  Nukaya, Manabu ^a,b   Walisser, Jacqueline A ^a   Moran, Susan M ^a   Kennedy, Gregory D ^b   Bradfield, Christopher A ^a  

^a UNIVERSITY OF WISCONSIN MADISON   (United States)

^b UNIVERSITY OF WISCONSIN SCHOOL OF MEDICINE AND PUBLIC HEALTH   (United States)

Author keywords

AHR; ARNT; Dioxin; Hepatocyte; Liver toxicity

Indexed keywords

AROMATIC HYDROCARBON RECEPTOR; DIOXIN; HETERODIMER; HYPOXIA INDUCIBLE FACTOR; HYPOXIA INDUCIBLE FACTOR 1BETA;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; FEMALE; LIVER CELL; LIVER CIRCULATION; LIVER TOXICITY; MALE; MOUSE; NONHUMAN; PROTEIN FUNCTION; VASCULARIZATION;

ADAPTATION, PHYSIOLOGICAL; ANIMALS; ARYL HYDROCARBON RECEPTOR NUCLEAR TRANSLOCATOR; DRUG-INDUCED LIVER INJURY; ENVIRONMENTAL POLLUTANTS; GENE DELETION; GENE EXPRESSION; GENE TARGETING; HEPATOCYTES; INJECTIONS, INTRAPERITONEAL; LIVER; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; RECEPTORS, ARYL HYDROCARBON; TETRACHLORODIBENZODIOXIN;

EID: 78549278788     PISSN: 10966080     EISSN: 10960929     Source Type: Journal    
DOI: 10.1093/toxsci/kfq305     Document Type: Article

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