Conditional overexpression of neuronal nitric oxide synthase is cardioprotective in ischemia/reperfusion

Circulation

Volumn 122, Issue 16, 2010, Pages 1588-1603

  Burkard, Natalie ^a   Williams, Tatjana ^a   Czolbe, Martin ^a   Blömer, Nadja ^a   Panther, Franziska ^a   Link, Martin ^a   Fraccarollo, Daniela ^a   Widder, Julian D ^a   Hu, Kai ^a   Han, Hong ^a   Hofmann, Ulrich ^a   Frantz, Stefan ^a   Nordbeck, Peter ^a   Bulla, Jan ^b   Schuh, Kai ^a   Ritter, Oliver ^a  

^a UNIVERSITY OF WÜRZBURG   (Germany)

^b UNIVERSITÉ DE CAEN   (France)

Author keywords

ischemia; mitochondria; nitric oxide synthase; reperfusion

Indexed keywords

HEAT SHOCK PROTEIN 90; NEURONAL NITRIC OXIDE SYNTHASE; NITRITE; REACTIVE OXYGEN METABOLITE; CYTOCHROME C OXIDASE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL FUNCTION; CONTROLLED STUDY; ENZYME ACTIVITY; FEMALE; HEART INFARCTION; HEART MUSCLE CELL; HEART MUSCLE OXYGEN CONSUMPTION; HEART PROTECTION; IN VIVO STUDY; ISOLATED HEART; MALE; MITOCHONDRION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; ANIMAL; GENETICS; HEART LEFT VENTRICLE FUNCTION; HEART MITOCHONDRION; METABOLISM; OXYGEN CONSUMPTION; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; REPERFUSION INJURY; TRANSGENIC MOUSE;

ANIMALS; ELECTRON TRANSPORT COMPLEX IV; FEMALE; MALE; MICE; MICE, TRANSGENIC; MITOCHONDRIA, HEART; MODELS, ANIMAL; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; NADPH OXIDASE; NITRIC OXIDE SYNTHASE TYPE I; OXYGEN CONSUMPTION; REACTIVE OXYGEN SPECIES; VENTRICULAR DYSFUNCTION, LEFT;

EID: 78049506757     PISSN: 00097322     EISSN: 15244539     Source Type: Journal    
DOI: 10.1161/CIRCULATIONAHA.109.933630     Document Type: Article

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