Deficiency of neuronal nitric oxide synthase increases mortality and cardiac remodeling after myocardial infarction: Role of nitroso-redox equilibrium

Circulation

Volumn 112, Issue 22, 2005, Pages 3415-3422

  Saraiva, Roberto M ^a,b   Minhas, Khalid M ^a   Raju, Shubha V Y ^a   Barouch, Lili A ^a   Pitz, Eleanor ^a   Schuleri, Karl H ^a   Vandegaer, Koenraad ^a   Li, Dechun ^a   Hare, Joshua M ^a  

^a DEPARTMENT OF PATHOLOGY   (United States)

^b FEDERAL UNIVERSITY OF SÃO PAULO   (Brazil)

Author keywords

Heart failure; Myocardial infarction; Nitric oxide synthase; Receptors, adrenergic, beta

Indexed keywords

NEURONAL NITRIC OXIDE SYNTHASE; NITRIC OXIDE; NITROSO DERIVATIVE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE; SUPEROXIDE; XANTHINE OXIDASE;

ADRENERGIC SYSTEM; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL MEMBRANE; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME DEFICIENCY; ENZYME LOCALIZATION; EXCITATION CONTRACTION COUPLING; FEMALE; HEART INFARCTION; HEART LEFT VENTRICLE FILLING; HEART MUSCLE CONTRACTILITY; HEART MUSCLE INJURY; HEART PROTECTION; MALE; MORTALITY; MOUSE; NEURONAL NITRIC OXIDE SYNTHASE DEFICIENCY; NONHUMAN; OXIDATION REDUCTION POTENTIAL; OXIDATIVE STRESS; PRIORITY JOURNAL; SARCOPLASMIC RETICULUM; SIGNAL TRANSDUCTION; ANIMAL; CARDIOMEGALY; ENZYMOLOGY; HEART LEFT VENTRICLE FUNCTION; HOMEOSTASIS; METABOLISM; MOUSE MUTANT; OXIDATION REDUCTION REACTION; PHYSIOLOGY;

ANIMALS; CARDIOMEGALY; HOMEOSTASIS; MICE; MICE, KNOCKOUT; MORTALITY; MYOCARDIAL INFARCTION; NADPH OXIDASE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE TYPE I; OXIDATION-REDUCTION; OXIDATIVE STRESS; SUPEROXIDES; VENTRICULAR DYSFUNCTION, LEFT;

EID: 33644873609     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/CIRCULATIONAHA.105.557892     Document Type: Article

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