-
1
-
-
0036360019
-
The enemy within: Keeping self-reactive T cells at bay in the periphery
-
Walker, L. S., and A. K. Abbas. 2002. The enemy within: keeping self-reactive T cells at bay in the periphery. Nat. Rev. Immunol. 2: 11-19.
-
(2002)
Nat. Rev. Immunol.
, vol.2
, pp. 11-19
-
-
Walker, L.S.1
Abbas, A.K.2
-
2
-
-
0029978038
-
Checkpoints in the progression of autoimmune disease: Lessons from diabetes models
-
André, I., A. Gonzalez, B. Wang, J. Katz, C. Benoist, and D. Mathis. 1996. Checkpoints in the progression of autoimmune disease: lessons from diabetes models. Proc. Natl. Acad. Sci. USA 93: 2260-2263.
-
(1996)
Proc. Natl. Acad. Sci. USA
, vol.93
, pp. 2260-2263
-
-
André, I.1
Gonzalez, A.2
Wang, B.3
Katz, J.4
Benoist, C.5
Mathis, D.6
-
3
-
-
2642655282
-
Cytotoxic T lymphocyte-associated antigen 4 (CTLA-4) regulates the unfolding of autoimmune diabetes
-
Lühder, F., P. Höglund, J. P. Allison, C. Benoist, and D. Mathis. 1998. Cytotoxic T lymphocyte-associated antigen 4 (CTLA-4) regulates the unfolding of autoimmune diabetes. J. Exp. Med. 187: 427-432.
-
(1998)
J. Exp. Med.
, vol.187
, pp. 427-432
-
-
Lühder, F.1
Höglund, P.2
Allison, J.P.3
Benoist, C.4
Mathis, D.5
-
4
-
-
0036884729
-
CTLA-4 differentially regulates T cell responses to endogenous tissue protein versus exogenous immunogen
-
Walker, L. S., L. J. Ausubel, A. Chodos, N. Bekarian, and A. K. Abbas. 2002. CTLA-4 differentially regulates T cell responses to endogenous tissue protein versus exogenous immunogen. J. Immunol. 169: 6202-6209.
-
(2002)
J. Immunol.
, vol.169
, pp. 6202-6209
-
-
Walker, L.S.1
Ausubel, L.J.2
Chodos, A.3
Bekarian, N.4
Abbas, A.K.5
-
6
-
-
0033579875
-
Initiation of autoimmune diabetes by developmentally regulated presentation of islet cell antigens in the pancreatic lymph nodes
-
Höglund, P., J. Mintern, C. Waltzinger, W. Heath, C. Benoist, and D. Mathis. 1999. Initiation of autoimmune diabetes by developmentally regulated presentation of islet cell antigens in the pancreatic lymph nodes. J. Exp. Med. 189: 331-339.
-
(1999)
J. Exp. Med.
, vol.189
, pp. 331-339
-
-
Höglund, P.1
Mintern, J.2
Waltzinger, C.3
Heath, W.4
Benoist, C.5
Mathis, D.6
-
7
-
-
33746932816
-
An inflammatory checkpoint regulates recruitment of graft-versus-host reactive T cells to peripheral tissues
-
Chakraverty, R., D. Côté, J. Buchli, P. Cotter, R. Hsu, G. Zhao, T. Sachs, C. M. Pitsillides, R. Bronson, T. Means, et al. 2006. An inflammatory checkpoint regulates recruitment of graft-versus-host reactive T cells to peripheral tissues. J. Exp. Med. 203: 2021-2031.
-
(2006)
J. Exp. Med.
, vol.203
, pp. 2021-2031
-
-
Chakraverty, R.1
Côté, D.2
Buchli, J.3
Cotter, P.4
Hsu, R.5
Zhao, G.6
Sachs, T.7
Pitsillides, C.M.8
Bronson, R.9
Means, T.10
-
8
-
-
0024312829
-
The natural history of lymphocyte subsets infiltrating the pancreas of NOD mice
-
Signore, A., P. Pozzilli, E. A. Gale, D. Andreani, and P. C. Beverley. 1989. The natural history of lymphocyte subsets infiltrating the pancreas of NOD mice. Diabetologia 32: 282-289.
-
(1989)
Diabetologia
, vol.32
, pp. 282-289
-
-
Signore, A.1
Pozzilli, P.2
Gale, E.A.3
Andreani, D.4
Beverley, P.C.5
-
9
-
-
0025553819
-
Flow cytometric enumeration of mononuclear cell populations infiltrating the islets of Langerhans in prediabetic NOD mice: Development of a model of autoimmune insulitis for type I diabetes
-
Jarpe, A. J., M. R. Hickman, J. T. Anderson, W. E. Winter, and A. B. Peck. 1990-1991. Flow cytometric enumeration of mononuclear cell populations infiltrating the islets of Langerhans in prediabetic NOD mice: development of a model of autoimmune insulitis for type I diabetes. Reg. Immunol. 3: 305-317.
-
(1990)
Reg. Immunol.
, vol.3
, pp. 305-317
-
-
Jarpe, A.J.1
Hickman, M.R.2
Anderson, J.T.3
Winter, W.E.4
Peck, A.B.5
-
10
-
-
0021799841
-
Prediabetes in the spontaneously diabetic BB/E rat: Lymphocyte subpopulations in the pancreatic infiltrate and expression of rat MHC class II molecules in endocrine cells
-
Dean, B. M., R. Walker, A. J. Bone, J. D. Baird, and A. Cooke. 1985. Prediabetes in the spontaneously diabetic BB/E rat: lymphocyte subpopulations in the pancreatic infiltrate and expression of rat MHC class II molecules in endocrine cells. Diabetologia 28: 464-466.
-
(1985)
Diabetologia
, vol.28
, pp. 464-466
-
-
Dean, B.M.1
Walker, R.2
Bone, A.J.3
Baird, J.D.4
Cooke, A.5
-
11
-
-
45949090105
-
Release from regulatory T cell-mediated suppression during the onset of tissue-specific autoimmunity is associated with elevated IL-21
-
Clough, L. E., C. J. Wang, E. M. Schmidt, G. Booth, T. Z. Hou, G. A. Ryan, and L. S. Walker. 2008. Release from regulatory T cell-mediated suppression during the onset of tissue-specific autoimmunity is associated with elevated IL-21. J. Immunol. 180: 5393-5401.
-
(2008)
J. Immunol.
, vol.180
, pp. 5393-5401
-
-
Clough, L.E.1
Wang, C.J.2
Schmidt, E.M.3
Booth, G.4
Hou, T.Z.5
Ryan, G.A.6
Walker, L.S.7
-
12
-
-
58149299735
-
Analysis of islet inflammation in human type 1 diabetes
-
Willcox, A., S. J. Richardson, A. J. Bone, A. K. Foulis, and N. G. Morgan. 2009. Analysis of islet inflammation in human type 1 diabetes. Clin. Exp. Immunol. 155: 173-181.
-
(2009)
Clin. Exp. Immunol.
, vol.155
, pp. 173-181
-
-
Willcox, A.1
Richardson, S.J.2
Bone, A.J.3
Foulis, A.K.4
Morgan, N.G.5
-
13
-
-
0035085973
-
Development and function of diabetogenic T-cells in B-cell-deficient nonobese diabetic mice
-
Chiu, P. P., D. V. Serreze, and J. S. Danska. 2001. Development and function of diabetogenic T-cells in B-cell-deficient nonobese diabetic mice. Diabetes 50: 763-770.
-
(2001)
Diabetes
, vol.50
, pp. 763-770
-
-
Chiu, P.P.1
Serreze, D.V.2
Danska, J.S.3
-
14
-
-
0035807589
-
Development of type 1 diabetes despite severe hereditary B-lymphocyte deficiency
-
Martin, S., D. Wolf-Eichbaum, G. Duinkerken, W. A. Scherbaum, H. Kolb, J. G. Noordzij, and B. O. Roep. 2001. Development of type 1 diabetes despite severe hereditary B-lymphocyte deficiency. N. Engl. J. Med. 345: 1036-1040.
-
(2001)
N. Engl. J. Med.
, vol.345
, pp. 1036-1040
-
-
Martin, S.1
Wolf-Eichbaum, D.2
Duinkerken, G.3
Scherbaum, W.A.4
Kolb, H.5
Noordzij, J.G.6
Roep, B.O.7
-
15
-
-
0029826977
-
B lymphocytes are essential for the initiation of T cell-mediated autoimmune diabetes: Analysis of a new "speed congenic" stock of NOD.Ig m null mice
-
Serreze, D. V., H. D. Chapman, D. S. Varnum, M. S. Hanson, P. C. Reifsnyder, S. D. Richard, S. A. Fleming, E. H. Leiter, and L. D. Shultz. 1996. B lymphocytes are essential for the initiation of T cell-mediated autoimmune diabetes: analysis of a new "speed congenic" stock of NOD.Ig m null mice. J. Exp. Med. 184: 2049-2053.
-
(1996)
J. Exp. Med.
, vol.184
, pp. 2049-2053
-
-
Serreze, D.V.1
Chapman, H.D.2
Varnum, D.S.3
Hanson, M.S.4
Reifsnyder, P.C.5
Richard, S.D.6
Fleming, S.A.7
Leiter, E.H.8
Shultz, L.D.9
-
16
-
-
8544271680
-
Direct evidence for the contribution of B cells to the progression of insulitis and the development of diabetes in non-obese diabetic mice
-
Akashi, T., S. Nagafuchi, K. Anzai, S. Kondo, D. Kitamura, S. Wakana, J. Ono, M. Kikuchi, Y. Niho, and T. Watanabe. 1997. Direct evidence for the contribution of B cells to the progression of insulitis and the development of diabetes in non-obese diabetic mice. Int. Immunol. 9: 1159-1164.
-
(1997)
Int. Immunol.
, vol.9
, pp. 1159-1164
-
-
Akashi, T.1
Nagafuchi, S.2
Anzai, K.3
Kondo, S.4
Kitamura, D.5
Wakana, S.6
Ono, J.7
Kikuchi, M.8
Niho, Y.9
Watanabe, T.10
-
17
-
-
0035887763
-
Impaired activation of islet-reactive CD4 T cells in pancreatic lymph nodes of B cell-deficient nonobese diabetic mice
-
Greeley, S. A., D. J. Moore, H. Noorchashm, L. E. Noto, S. Y. Rostami, A. Schlachterman, H. K. Song, B. Koeberlein, C. F. Barker, and A. Naji. 2001. Impaired activation of islet-reactive CD4 T cells in pancreatic lymph nodes of B cell-deficient nonobese diabetic mice. J. Immunol. 167: 4351-4357.
-
(2001)
J. Immunol.
, vol.167
, pp. 4351-4357
-
-
Greeley, S.A.1
Moore, D.J.2
Noorchashm, H.3
Noto, L.E.4
Rostami, S.Y.5
Schlachterman, A.6
Song, H.K.7
Koeberlein, B.8
Barker, C.F.9
Naji, A.10
-
18
-
-
0030969531
-
B-cells are required for the initiation of insulitis and sialitis in nonobese diabetic mice
-
Noorchashm, H., N. Noorchashm, J. Kern, S. Y. Rostami, C. F. Barker, and A. Naji. 1997. B-cells are required for the initiation of insulitis and sialitis in nonobese diabetic mice. Diabetes 46: 941-946.
-
(1997)
Diabetes
, vol.46
, pp. 941-946
-
-
Noorchashm, H.1
Noorchashm, N.2
Kern, J.3
Rostami, S.Y.4
Barker, C.F.5
Naji, A.6
-
19
-
-
0034652317
-
Early expression of anti-insulin autoantibodies of humans and the NOD mouse: Evidence for early determination of subsequent diabetes
-
Yu, L., D. T. Robles, N. Abiru, P. Kaur, M. Rewers, K. Kelemen, and G. S. Eisenbarth. 2000. Early expression of anti-insulin autoantibodies of humans and the NOD mouse: evidence for early determination of subsequent diabetes. Proc. Natl. Acad. Sci. USA 97: 1701-1706.
-
(2000)
Proc. Natl. Acad. Sci. USA
, vol.97
, pp. 1701-1706
-
-
Yu, L.1
Robles, D.T.2
Abiru, N.3
Kaur, P.4
Rewers, M.5
Kelemen, K.6
Eisenbarth, G.S.7
-
20
-
-
4644225746
-
Investigation of the role of B-cells in type 1 diabetes in the NOD mouse
-
Wong, F. S., L. Wen, M. Tang, M. Ramanathan, I. Visintin, J. Daugherty, L. G. Hannum, C. A. Janeway, Jr., and M. J. Shlomchik. 2004. Investigation of the role of B-cells in type 1 diabetes in the NOD mouse. Diabetes 53: 2581-2587.
-
(2004)
Diabetes
, vol.53
, pp. 2581-2587
-
-
Wong, F.S.1
Wen, L.2
Tang, M.3
Ramanathan, M.4
Visintin, I.5
Daugherty, J.6
Hannum, L.G.7
Janeway Jr., C.A.8
Shlomchik, M.J.9
-
21
-
-
0032532294
-
B lymphocytes are critical antigen-presenting cells for the initiation of T cell-mediated autoimmune diabetes in nonobese diabetic mice
-
Serreze, D. V., S. A. Fleming, H. D. Chapman, S. D. Richard, E. H. Leiter, and R. M. Tisch. 1998. B lymphocytes are critical antigen-presenting cells for the initiation of T cell-mediated autoimmune diabetes in nonobese diabetic mice. J. Immunol. 161: 3912-3918.
-
(1998)
J. Immunol.
, vol.161
, pp. 3912-3918
-
-
Serreze, D.V.1
Fleming, S.A.2
Chapman, H.D.3
Richard, S.D.4
Leiter, E.H.5
Tisch, R.M.6
-
22
-
-
0032145276
-
B lymphocytes are crucial antigen-presenting cells in the pathogenic autoimmune response to GAD65 antigen in nonobese diabetic mice
-
Falcone, M., J. Lee, G. Patstone, B. Yeung, and N. Sarvetnick. 1998. B lymphocytes are crucial antigen-presenting cells in the pathogenic autoimmune response to GAD65 antigen in nonobese diabetic mice. J. Immunol. 161: 1163-1168.
-
(1998)
J. Immunol.
, vol.161
, pp. 1163-1168
-
-
Falcone, M.1
Lee, J.2
Patstone, G.3
Yeung, B.4
Sarvetnick, N.5
-
24
-
-
70449480577
-
Rituximab, B-lymphocyte depletion, and preservation of β-cell function
-
Pescovitz, M. D., C. J. Greenbaum, H. Krause-Steinrauf, D. J. Becker, S. E. Gitelman, R. Goland, P. A. Gottlieb, J. B. Marks, P. F. McGee, A. M. Moran, et al. 2009. Rituximab, B-lymphocyte depletion, and preservation of β-cell function. N. Engl. J. Med. 361: 2143-2152.
-
(2009)
N. Engl. J. Med.
, vol.361
, pp. 2143-2152
-
-
Pescovitz, M.D.1
Greenbaum, C.J.2
Krause-Steinrauf, H.3
Becker, D.J.4
Gitelman, S.E.5
Goland, R.6
Gottlieb, P.A.7
Marks, J.B.8
McGee, P.F.9
Moran, A.M.10
-
25
-
-
36849027817
-
Treatment with CD20-specific antibody prevents and reverses autoimmune diabetes in mice
-
Hu, C. Y., D. Rodriguez-Pinto, W. Du, A. Ahuja, O. Henegariu, F. S. Wong, M. J. Shlomchik, and L. Wen. 2007. Treatment with CD20-specific antibody prevents and reverses autoimmune diabetes in mice. J. Clin. Invest. 117: 3857-3867.
-
(2007)
J. Clin. Invest.
, vol.117
, pp. 3857-3867
-
-
Hu, C.Y.1
Rodriguez-Pinto, D.2
Du, W.3
Ahuja, A.4
Henegariu, O.5
Wong, F.S.6
Shlomchik, M.J.7
Wen, L.8
-
26
-
-
58149359257
-
Targeting CD22 reprograms B-cells and reverses autoimmune diabetes
-
Fiorina, P., A. Vergani, S. Dada, M. Jurewicz, M. Wong, K. Law, E. Wu, Z. Tian, R. Abdi, I. Guleria, et al. 2008. Targeting CD22 reprograms B-cells and reverses autoimmune diabetes. Diabetes 57: 3013-3024.
-
(2008)
Diabetes
, vol.57
, pp. 3013-3024
-
-
Fiorina, P.1
Vergani, A.2
Dada, S.3
Jurewicz, M.4
Wong, M.5
Law, K.6
Wu, E.7
Tian, Z.8
Abdi, R.9
Guleria, I.10
-
27
-
-
0028102655
-
High incidence of spontaneous autoimmune encephalomyelitis in immunodeficient antimyelin basic protein T cell receptor transgenic mice
-
Lafaille, J. J., K. Nagashima, M. Katsuki, and S. Tonegawa. 1994. High incidence of spontaneous autoimmune encephalomyelitis in immunodeficient antimyelin basic protein T cell receptor transgenic mice. Cell 78: 399-408.
-
(1994)
Cell
, vol.78
, pp. 399-408
-
-
Lafaille, J.J.1
Nagashima, K.2
Katsuki, M.3
Tonegawa, S.4
-
30
-
-
27144474948
-
B-cell depletion inhibits arthritis in a collagen-induced arthritis (CIA) model, but does not adversely affect humoral responses in a respiratory syncytial virus (RSV) vaccination model
-
Dunussi-Joannopoulos, K., G. E. Hancock, A. Kunz, M. Hegen, X. X. Zhou, B. J. Sheppard, J. Lamothe, E. Li, H. L. Ma, P. R. Hamann, et al. 2005. B-cell depletion inhibits arthritis in a collagen-induced arthritis (CIA) model, but does not adversely affect humoral responses in a respiratory syncytial virus (RSV) vaccination model. Blood 106: 2235-2243.
-
(2005)
Blood
, vol.106
, pp. 2235-2243
-
-
Dunussi-Joannopoulos, K.1
Hancock, G.E.2
Kunz, A.3
Hegen, M.4
Zhou, X.X.5
Sheppard, B.J.6
Lamothe, J.7
Li, E.8
Ma, H.L.9
Hamann, P.R.10
-
32
-
-
0032560571
-
Transfer of human serum IgG to nonobese diabetic Igμ null mice reveals a role for autoantibodies in the loss of secretory function of exocrine tissues in Sjögren's syndrome
-
Robinson, C. P., J. Brayer, S. Yamachika, T. R. Esch, A. B. Peck, C. A. Stewart, E. Peen, R. Jonsson, and M. G. Humphreys-Beher. 1998. Transfer of human serum IgG to nonobese diabetic Igμ null mice reveals a role for autoantibodies in the loss of secretory function of exocrine tissues in Sjögren's syndrome. Proc. Natl. Acad. Sci. USA 95: 7538-7543.
-
(1998)
Proc. Natl. Acad. Sci. USA
, vol.95
, pp. 7538-7543
-
-
Robinson, C.P.1
Brayer, J.2
Yamachika, S.3
Esch, T.R.4
Peck, A.B.5
Stewart, C.A.6
Peen, E.7
Jonsson, R.8
Humphreys-Beher, M.G.9
-
33
-
-
0033118675
-
From systemic T cell self-reactivity to organ-specific autoimmune disease via immunoglobulins
-
Korganow, A. S., H. Ji, S. Mangialaio, V. Duchatelle, R. Pelanda, T. Martin, C. Degott, H. Kikutani, K. Rajewsky, J. L. Pasquali, et al. 1999. From systemic T cell self-reactivity to organ-specific autoimmune disease via immunoglobulins. Immunity 10: 451-461.
-
(1999)
Immunity
, vol.10
, pp. 451-461
-
-
Korganow, A.S.1
Ji, H.2
Mangialaio, S.3
Duchatelle, V.4
Pelanda, R.5
Martin, T.6
Degott, C.7
Kikutani, H.8
Rajewsky, K.9
Pasquali, J.L.10
-
34
-
-
0345448056
-
Physiological β cell death triggers priming of self-reactive T cells by dendritic cells in a type-1-diabetes model
-
Turley, S., L. Poirot, M. Hattori, C. Benoist, and D. Mathis. 2003. Physiological β cell death triggers priming of self-reactive T cells by dendritic cells in a type-1-diabetes model. J. Exp. Med. 198: 1527-1537.
-
(2003)
J. Exp. Med.
, vol.198
, pp. 1527-1537
-
-
Turley, S.1
Poirot, L.2
Hattori, M.3
Benoist, C.4
Mathis, D.5
-
35
-
-
0027207669
-
Transgenic tumor necrosis factor (TNF)-α production in pancreatic islets leads to insulitis, not diabetes: Distinct patterns of inflammation in TNF-α and TNF-β transgenic mice
-
Picarella, D. E., A. Kratz, C. B. Li, N. H. Ruddle, and R. A. Flavell. 1993. Transgenic tumor necrosis factor (TNF)-α production in pancreatic islets leads to insulitis, not diabetes: distinct patterns of inflammation in TNF-α and TNF-β transgenic mice. J. Immunol. 150: 4136-4150.
-
(1993)
J. Immunol.
, vol.150
, pp. 4136-4150
-
-
Picarella, D.E.1
Kratz, A.2
Li, C.B.3
Ruddle, N.H.4
Flavell, R.A.5
-
36
-
-
33847058565
-
Islet β-cell-specific T cells can use different homing mechanisms to infiltrate and destroy pancreatic islets
-
Hänninen, A., R. Nurmela, M. Maksimow, J. Heino, S. Jalkanen, and C. Kurts. 2007. Islet β-cell-specific T cells can use different homing mechanisms to infiltrate and destroy pancreatic islets. Am. J. Pathol. 170: 240-250.
-
(2007)
Am. J. Pathol.
, vol.170
, pp. 240-250
-
-
Hänninen, A.1
Nurmela, R.2
Maksimow, M.3
Heino, J.4
Jalkanen, S.5
Kurts, C.6
-
37
-
-
0036883945
-
The natural autoantibody repertoire of nonobese diabetic mice is highly active
-
Thomas, J. W., P. L. Kendall, and H. G. Mitchell. 2002. The natural autoantibody repertoire of nonobese diabetic mice is highly active. J. Immunol. 169: 6617-6624.
-
(2002)
J. Immunol.
, vol.169
, pp. 6617-6624
-
-
Thomas, J.W.1
Kendall, P.L.2
Mitchell, H.G.3
-
38
-
-
77954232248
-
Reduced diabetes in btk-deficient nonobese diabetic mice and restoration of diabetes with provision of an anti-insulin IgH chain transgene
-
Kendall, P. L., D. J. Moore, C. Hulbert, K. L. Hoek, W. N. Khan, and J. W. Thomas. 2009. Reduced diabetes in btk-deficient nonobese diabetic mice and restoration of diabetes with provision of an anti-insulin IgH chain transgene. J. Immunol. 183: 6403-6412.
-
(2009)
J. Immunol.
, vol.183
, pp. 6403-6412
-
-
Kendall, P.L.1
Moore, D.J.2
Hulbert, C.3
Hoek, K.L.4
Khan, W.N.5
Thomas, J.W.6
-
39
-
-
4644310776
-
Peritoneal B cells govern the outcome of diabetes in non-obese diabetic mice
-
Kendall, P. L., E. J. Woodward, C. Hulbert, and J. W. Thomas. 2004. Peritoneal B cells govern the outcome of diabetes in non-obese diabetic mice. Eur. J. Immunol. 34: 2387-2395.
-
(2004)
Eur. J. Immunol.
, vol.34
, pp. 2387-2395
-
-
Kendall, P.L.1
Woodward, E.J.2
Hulbert, C.3
Thomas, J.W.4
-
40
-
-
11844290756
-
Dysregulated B7-1 and B7-2 expression on nonobese diabetic mouse B cells is associated with increased T cell costimulation and the development of insulitis
-
Hussain, S., and T. L. Delovitch. 2005. Dysregulated B7-1 and B7-2 expression on nonobese diabetic mouse B cells is associated with increased T cell costimulation and the development of insulitis. J. Immunol. 174: 680-687.
-
(2005)
J. Immunol.
, vol.174
, pp. 680-687
-
-
Hussain, S.1
Delovitch, T.L.2
-
41
-
-
34250179766
-
Novel suppressive function of transitional 2 B cells in experimental arthritis
-
Evans, J. G., K. A. Chavez-Rueda, A. Eddaoudi, A. Meyer-Bahlburg, D. J. Rawlings, M. R. Ehrenstein, and C. Mauri. 2007. Novel suppressive function of transitional 2 B cells in experimental arthritis. J. Immunol. 178: 7868-7878.
-
(2007)
J. Immunol.
, vol.178
, pp. 7868-7878
-
-
Evans, J.G.1
Chavez-Rueda, K.A.2
Eddaoudi, A.3
Meyer-Bahlburg, A.4
Rawlings, D.J.5
Ehrenstein, M.R.6
Mauri, C.7
-
43
-
-
77951640991
-
Cutting edge: Novel function of B cell-activating factor in the induction of IL-10-producing regulatory B cells
-
Yang, M., L. Sun, S. Wang, K. H. Ko, H. Xu, B. J. Zheng, X. Cao, and L. Lu. 2010. Cutting edge: novel function of B cell-activating factor in the induction of IL-10-producing regulatory B cells. J. Immunol. 184: 3321-3325.
-
(2010)
J. Immunol.
, vol.184
, pp. 3321-3325
-
-
Yang, M.1
Sun, L.2
Wang, S.3
Ko, K.H.4
Xu, H.5
Zheng, B.J.6
Cao, X.7
Lu, L.8
-
44
-
-
0036795531
-
B cells regulate autoimmunity by provision of IL-10
-
Fillatreau, S., C. H. Sweenie, M. J. McGeachy, D. Gray, and S. M. Anderton. 2002. B cells regulate autoimmunity by provision of IL-10. Nat. Immunol. 3: 944-950.
-
(2002)
Nat. Immunol.
, vol.3
, pp. 944-950
-
-
Fillatreau, S.1
Sweenie, C.H.2
McGeachy, M.J.3
Gray, D.4
Anderton, S.M.5
-
45
-
-
0036197923
-
Chronic intestinal inflammatory condition generates IL-10-producing regulatory B cell subset characterized by CD1d upregulation
-
Mizoguchi, A., E. Mizoguchi, H. Takedatsu, R. S. Blumberg, and A. K. Bhan. 2002. Chronic intestinal inflammatory condition generates IL-10-producing regulatory B cell subset characterized by CD1d upregulation. Immunity 16: 219-230.
-
(2002)
Immunity
, vol.16
, pp. 219-230
-
-
Mizoguchi, A.1
Mizoguchi, E.2
Takedatsu, H.3
Blumberg, R.S.4
Bhan, A.K.5
-
46
-
-
51349138423
-
B cells are required for Aire-deficient mice to develop multi-organ autoinflammation: A therapeutic approach for APECED patients
-
Gavanescu, I., C. Benoist, and D. Mathis. 2008. B cells are required for Aire-deficient mice to develop multi-organ autoinflammation: a therapeutic approach for APECED patients. Proc. Natl. Acad. Sci. USA 105: 13009-13014.
-
(2008)
Proc. Natl. Acad. Sci. USA
, vol.105
, pp. 13009-13014
-
-
Gavanescu, I.1
Benoist, C.2
Mathis, D.3
-
47
-
-
70449495408
-
Novel targeted therapies for autoimmunity
-
St. Clair, E. W. 2009. Novel targeted therapies for autoimmunity. Curr. Opin. Immunol. 21: 648-657.
-
(2009)
Curr. Opin. Immunol.
, vol.21
, pp. 648-657
-
-
St Clair, E.W.1
-
48
-
-
0026506870
-
Ly-1 B (B-1) cells are the main source of B cell-derived interleukin 10
-
O'Garra, A., R. Chang, N. Go, R. Hastings, G. Haughton, and M. Howard. 1992. Ly-1 B (B-1) cells are the main source of B cell-derived interleukin 10. Eur. J. Immunol. 22: 711-717.
-
(1992)
Eur. J. Immunol.
, vol.22
, pp. 711-717
-
-
O'Garra, A.1
Chang, R.2
Go, N.3
Hastings, R.4
Haughton, G.5
Howard, M.6
-
49
-
-
77949798304
-
An unexpected counter-regulatory role of IL-10 in B-lymphocyte-mediated transplantation tolerance
-
Zhao, G., D. J. Moore, K. M. Lee, J. I. Kim, P. E. Duff, M. R. O'Connor, T. Hirohashi, J. Lei, M. Yang, J. F. Markmann, and S. Deng. 2010. An unexpected counter-regulatory role of IL-10 in B-lymphocyte-mediated transplantation tolerance. Am. J. Transplant. 10: 796-801.
-
(2010)
Am. J. Transplant.
, vol.10
, pp. 796-801
-
-
Zhao, G.1
Moore, D.J.2
Lee, K.M.3
Kim, J.I.4
Duff, P.E.5
O'Connor, M.R.6
Hirohashi, T.7
Lei, J.8
Yang, M.9
Markmann, J.F.10
Deng, S.11
-
50
-
-
0028346888
-
Production of interleukin 10 by islet cells accelerates immune-mediated destruction of b cells in nonobese diabetic mice
-
Wogensen, L., M. S. Lee, and N. Sarvetnick. 1994. Production of interleukin 10 by islet cells accelerates immune-mediated destruction of b cells in nonobese diabetic mice. J. Exp. Med. 179: 1379-1384.
-
(1994)
J. Exp. Med.
, vol.179
, pp. 1379-1384
-
-
Wogensen, L.1
Lee, M.S.2
Sarvetnick, N.3
-
51
-
-
0034284446
-
Islet-specific expression of IL-10 promotes diabetes in nonobese diabetic mice independent of Fas, perforin, TNF receptor-1, and TNF receptor-2 molecules
-
Balasa, B., K. Van Gunst, N. Jung, D. Balakrishna, P. Santamaria, T. Hanafusa, N. Itoh, and N. Sarvetnick. 2000. Islet-specific expression of IL-10 promotes diabetes in nonobese diabetic mice independent of Fas, perforin, TNF receptor-1, and TNF receptor-2 molecules. J. Immunol. 165: 2841-2849.
-
(2000)
J. Immunol.
, vol.165
, pp. 2841-2849
-
-
Balasa, B.1
Van Gunst, K.2
Jung, N.3
Balakrishna, D.4
Santamaria, P.5
Hanafusa, T.6
Itoh, N.7
Sarvetnick, N.8
-
52
-
-
0031404443
-
Pancreatic IL-10 induces diabetes in NOD.B6 Idd3 Idd10 mice
-
Lee, M. S., L. S. Wicker, L. B. Peterson, and N. Sarvetnick. 1997. Pancreatic IL-10 induces diabetes in NOD.B6 Idd3 Idd10 mice. Autoimmunity 26: 215-221.
-
(1997)
Autoimmunity
, vol.26
, pp. 215-221
-
-
Lee, M.S.1
Wicker, L.S.2
Peterson, L.B.3
Sarvetnick, N.4
-
53
-
-
29144508394
-
Endocrine self and gut non-self intersect in the pancreatic lymph nodes
-
Turley, S. J., J. W. Lee, N. Dutton-Swain, D. Mathis, and C. Benoist. 2005. Endocrine self and gut non-self intersect in the pancreatic lymph nodes. Proc. Natl. Acad. Sci. USA 102: 17729-17733.
-
(2005)
Proc. Natl. Acad. Sci. USA
, vol.102
, pp. 17729-17733
-
-
Turley, S.J.1
Lee, J.W.2
Dutton-Swain, N.3
Mathis, D.4
Benoist, C.5
-
54
-
-
0347911963
-
Cutaneous immunization rapidly activates liver invariant Va14 NKT cells stimulating B-1 B cells to initiate T cell recruitment for elicitation of contact sensitivity
-
Campos, R. A., M. Szczepanik, A. Itakura, M. Akahira-Azuma, S. Sidobre, M. Kronenberg, and P. W. Askenase. 2003. Cutaneous immunization rapidly activates liver invariant Va14 NKT cells stimulating B-1 B cells to initiate T cell recruitment for elicitation of contact sensitivity. J. Exp. Med. 198: 1785-1796.
-
(2003)
J. Exp. Med.
, vol.198
, pp. 1785-1796
-
-
Campos, R.A.1
Szczepanik, M.2
Itakura, A.3
Akahira-Azuma, M.4
Sidobre, S.5
Kronenberg, M.6
Askenase, P.W.7
-
55
-
-
34447624878
-
B cell-specific deletion of protein-tyrosine phosphatase Shp1 promotes B-1a cell development and causes systemic autoimmunity
-
Pao, L. I., K. P. Lam, J. M. Henderson, J. L. Kutok, M. Alimzhanov, L. Nitschke, M. L. Thomas, B. G. Neel, and K. Rajewsky. 2007. B cell-specific deletion of protein-tyrosine phosphatase Shp1 promotes B-1a cell development and causes systemic autoimmunity. Immunity 27: 35-48.
-
(2007)
Immunity
, vol.27
, pp. 35-48
-
-
Pao, L.I.1
Lam, K.P.2
Henderson, J.M.3
Kutok, J.L.4
Alimzhanov, M.5
Nitschke, L.6
Thomas, M.L.7
Neel, B.G.8
Rajewsky, K.9
-
56
-
-
77951627428
-
CD22 X Siglec-G double-deficient mice have massively increased B1 cell numbers and develop systemic autoimmunity
-
Jellusova, J., U. Wellmann, K. Amann, T. H. Winkler, and L. Nitschke. 2010. CD22 X Siglec-G double-deficient mice have massively increased B1 cell numbers and develop systemic autoimmunity. J. Immunol. 184: 3618-3627.
-
(2010)
J. Immunol.
, vol.184
, pp. 3618-3627
-
-
Jellusova, J.1
Wellmann, U.2
Amann, K.3
Winkler, T.H.4
Nitschke, L.5
-
57
-
-
33750507279
-
Regulation of B1 cell migration by signals through Toll-like receptors
-
Ha, S. A., M. Tsuji, K. Suzuki, B. Meek, N. Yasuda, T. Kaisho, and S. Fagarasan. 2006. Regulation of B1 cell migration by signals through Toll-like receptors. J. Exp. Med. 203: 2541-2550.
-
(2006)
J. Exp. Med.
, vol.203
, pp. 2541-2550
-
-
Ha, S.A.1
Tsuji, M.2
Suzuki, K.3
Meek, B.4
Yasuda, N.5
Kaisho, T.6
Fagarasan, S.7
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