Prolonged Aβ treatment leads to impairment in the ability of primary cortical neurons to maintain K+ and Ca2+ homeostasis

Molecular Neurodegeneration

Volumn 5, Issue 1, 2010, Pages

  Shabala, Lana ^a   Howells, Claire ^a   West, Adrian K ^a   Chung, Roger S ^a  

^a UNIVERSITY OF TASMANIA   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN[1-40]; CALCIUM ION; GLUTAMIC ACID; POTASSIUM ION;

ACUTE TOXICITY; AGE; ALZHEIMER DISEASE; ANIMAL CELL; ANIMAL TISSUE; ARTICLE; BRAIN CELL; BRAIN CORTEX; CALCIUM TRANSPORT; CELL LEVEL; CELL SURVIVAL; CHRONIC DISEASE; CONTROLLED STUDY; CORRELATION ANALYSIS; DEGENERATIVE DISEASE; EMBRYO; FUTUROLOGY; HOMEOSTASIS; IN VITRO STUDY; LETHAL DOSE; LONG TERM EXPOSURE; NERVE CELL; NERVE CELL CULTURE; NEUROFIBRILLARY TANGLE; NEUROTOXICITY; NONHUMAN; PATHOGENESIS; POTASSIUM TRANSPORT; RAT; SURVIVAL; SYMPTOM; TREATMENT DURATION;

EID: 77955485267     PISSN: None     EISSN: 17501326     Source Type: Journal    
DOI: 10.1186/1750-1326-5-30     Document Type: Article

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