-
1
-
-
0035349906
-
The genetics and pathology of oxidative phosphorylation
-
Smeitink, J., L. Van Den Heuvel & S. DiMauro. 2001. The genetics and pathology of oxidative phosphorylation. Nat. Rev. Genet . 2: 342-352.
-
(2001)
Nat. Rev. Genet.
, vol.2
, pp. 342-352
-
-
Smeitink, J.1
Heuvel Den L.Van2
Dimauro, S.3
-
2
-
-
0033525773
-
Mitochondrial diseases in man and mouse
-
Wallace, D.C. 1999. Mitochondrial diseases in man and mouse. Science. 283: 1482-1488.
-
(1999)
Science
, vol.283
, pp. 1482-1488
-
-
Wallace, D.C.1
-
3
-
-
38949188752
-
Clinical and molecular features of mitochondrial DNA depletion due to mutations in deoxyguanosine kinase
-
Dimmock, D.P. et al. 2008. Clinical and molecular features of mitochondrial DNA depletion due to mutations in deoxyguanosine kinase. Hum Mutat. 29: 330-331.
-
(2008)
Hum Mutat
, vol.29
, pp. 330-331
-
-
Dimmock, D.P.1
-
4
-
-
36348966712
-
Mutations in the MPV17 gene are responsible for rapidly progressive liver failure in infancy
-
Wong, L.J. et al. 2007. Mutations in the MPV17 gene are responsible for rapidly progressive liver failure in infancy. Hepatology. 46: 1218-1227.
-
(2007)
Hepatology
, vol.46
, pp. 1218-1227
-
-
Wong, L.J.1
-
5
-
-
0034977818
-
Mitochondria: Integrators in tumorigenesis?
-
Augenlicht, L.H. & B.G. Heerdt. 2001. Mitochondria: integrators in tumorigenesis? Nat Genet . 28: 104-105.
-
(2001)
Nat Genet
, vol.28
, pp. 104-105
-
-
Augenlicht, L.H.1
Heerdt, B.G.2
-
6
-
-
38849175587
-
Mitochondrial cytochrome B gene mutation promotes tumor growth in bladder cancer
-
Dasgupta, S. et al. 2008. Mitochondrial cytochrome B gene mutation promotes tumor growth in bladder cancer. Cancer Res. 68: 700-706.
-
(2008)
Cancer Res
, vol.68
, pp. 700-706
-
-
Dasgupta, S.1
-
7
-
-
43249112094
-
ROS-generating mitochondrial DNA mutations can regulate tumor cell metastasis
-
Ishikawa,K. et al. 2008.ROS-generating mitochondrialDNA mutations can regulate tumor cell metastasis. Science. 320: 661-664.
-
(2008)
Science
, vol.320
, pp. 661-664
-
-
Ishikawa, K.1
-
8
-
-
43649083607
-
DNA polymerase gamma inhibition by vitamin K3 induces mitochondria-mediated cytotoxicity in human cancer cells
-
Sasaki, R. et al. 2008. DNA polymerase gamma inhibition by vitamin K3 induces mitochondria-mediated cytotoxicity in human cancer cells. Cancer Sci. 99: 1040-1048.
-
(2008)
Cancer Sci
, vol.99
, pp. 1040-1048
-
-
Sasaki, R.1
-
9
-
-
0030925951
-
Detection of somatic mutations in the mitochondrial DNA control region of colorectal and gastric tumors by heteroduplex and single-strand conformation analysis
-
Alonso, A. et al. 1997. Detection of somatic mutations in the mitochondrial DNA control region of colorectal and gastric tumors by heteroduplex and single-strand conformation analysis. Electrophoresis. 18: 682-685.
-
(1997)
Electrophoresis
, vol.18
, pp. 682-685
-
-
Alonso, A.1
-
10
-
-
0036293208
-
Mitochondrial DNA alterations in cancer
-
Copeland,W.C. et al. 2002. Mitochondrial DNA alterations in cancer. Cancer Invest. 20: 557-569.
-
(2002)
Cancer Invest
, vol.20
, pp. 557-569
-
-
Copeland, W.C.1
-
11
-
-
0039250954
-
Facile detection of mitochondrial DNA mutations in tumors and bodily fluids
-
Fliss, M.S. et al. 2000. Facile detection of mitochondrial DNA mutations in tumors and bodily fluids. Science. 287: 2017-2019.
-
(2000)
Science
, vol.287
, pp. 2017-2019
-
-
Fliss, M.S.1
-
12
-
-
0032729633
-
Mitochondrial gene mutation, but not large-scale deletion, is a feature of colorectal carcinomas with mitochondrial microsatellite instability
-
Habano, W. et al. 1999. Mitochondrial gene mutation, but not large-scale deletion, is a feature of colorectal carcinomas with mitochondrial microsatellite instability. Int. J. Cancer. 83: 625-629.
-
(1999)
Int. J. Cancer
, vol.83
, pp. 625-629
-
-
Habano, W.1
-
13
-
-
0035342502
-
Mitochondrial DNA alteration in esophageal cancer
-
Hibi, K. et al. 2001. Mitochondrial DNA alteration in esophageal cancer. Int. J. Cancer. 92: 319-321.
-
(2001)
Int. J. Cancer
, vol.92
, pp. 319-321
-
-
Hibi, K.1
-
14
-
-
0035882532
-
High frequency of mitochondrial DNA mutations in glioblastoma multiforme identified by direct sequence comparison to blood samples
-
Kirches, E. et al. 2001. High frequency of mitochondrial DNA mutations in glioblastoma multiforme identified by direct sequence comparison to blood samples. Int. J. Cancer. 93: 534-538.
-
(2001)
Int. J. Cancer
, vol.93
, pp. 534-538
-
-
Kirches, E.1
-
15
-
-
4344570901
-
SomaticmitochondrialDNAmutations in neurofibromatosis type 1-associated tumors
-
Kurtz, A. et al. 2004. SomaticmitochondrialDNAmutations in neurofibromatosis type 1-associated tumors. Mol. Cancer Res. 2: 433-441.
-
(2004)
Mol. Cancer Res
, vol.2
, pp. 433-441
-
-
Kurtz, A.1
-
16
-
-
0035882029
-
High incidence of somatic mitochondrial DNAmutations in human ovarian carcinomas
-
Liu, V.W. et al. 2001. High incidence of somatic mitochondrial DNAmutations in human ovarian carcinomas. Cancer Res. 61: 5998-6001.
-
(2001)
Cancer Res
, vol.61
, pp. 5998-6001
-
-
Liu, V.W.1
-
17
-
-
0031736203
-
Somatic mutations of the mitochondrial genome in human colorectal tumours
-
Polyak, K. et al. 1998. Somatic mutations of the mitochondrial genome in human colorectal tumours. Nat. Genet . 20: 291-293.
-
(1998)
Nat. Genet
, vol.20
, pp. 291-293
-
-
Polyak, K.1
-
18
-
-
0033083094
-
Mutations in mitochondrial control region DNA in gastric tumours of Japanese patients
-
Tamura, G. et al. 1999. Mutations in mitochondrial control region DNA in gastric tumours of Japanese patients. Eur. J. Cancer. 35: 316-319.
-
(1999)
Eur. J. Cancer
, vol.35
, pp. 316-319
-
-
Tamura, G.1
-
19
-
-
0036170578
-
Comprehensive scanning of somatic mitochondrial DNA mutations in breast cancer
-
Tan, D.J., R.K. Bai & L.J.Wong. 2002. Comprehensive scanning of somatic mitochondrial DNA mutations in breast cancer. Cancer Res. 62: 972-976.
-
(2002)
Cancer Res
, vol.62
, pp. 972-976
-
-
Tan, D.J.1
Bai, R.K.2
Wong, L.J.3
-
20
-
-
0038070419
-
Novel heteroplasmic frameshift andmissense somatic mitochondrial DNA mutations in oral cancer of betel quid chewers
-
Tan,D.J. et al. 2003.Novel heteroplasmic frameshift andmissense somatic mitochondrial DNA mutations in oral cancer of betel quid chewers. Genes Chromosomes Cancer. 37: 186-194.
-
(2003)
Genes Chromosomes Cancer
, vol.37
, pp. 186-194
-
-
Tan, D.J.1
-
21
-
-
33646559185
-
Significance of somatic mutations and content alteration ofmitochondrialDNA in esophageal cancer
-
Tan, D.J. et al. 2006. Significance of somatic mutations and content alteration ofmitochondrialDNA in esophageal cancer. BMC Cancer. 6: 93.
-
(2006)
BMC Cancer
, vol.6
, pp. 93
-
-
Tan, D.J.1
-
22
-
-
0041742269
-
Detection of mitochondrial DNA mutations in the tumor and cerebrospinal fluid of medulloblastoma patients
-
Wong, L.J. et al. 2003. Detection of mitochondrial DNA mutations in the tumor and cerebrospinal fluid of medulloblastoma patients. Cancer Res. 63: 3866-3871.
-
(2003)
Cancer Res
, vol.63
, pp. 3866-3871
-
-
Wong, L.J.1
-
23
-
-
3042766311
-
Molecular alterations in mitochondrial DNA of hepatocellular carcinomas: Is there a correlation with clinicopathological profile?
-
Wong, L.J. et al. 2004. Molecular alterations in mitochondrial DNA of hepatocellular carcinomas: is there a correlation with clinicopathological profile? J. Med. Genet . 41: e65.
-
(2004)
J. Med. Genet
, vol.41
-
-
Wong, L.J.1
-
24
-
-
0034643388
-
Somatic mitochondrial DNA (mtDNA) mutations in papillary thyroid carcinomas and differential mtDNA sequence variants in cases with thyroid tumours
-
Yeh, J.J. et al. 2000. Somatic mitochondrial DNA (mtDNA) mutations in papillary thyroid carcinomas and differential mtDNA sequence variants in cases with thyroid tumours. Oncogene 19: 2060-2066.
-
(2000)
Oncogene
, vol.19
, pp. 2060-2066
-
-
Yeh, J.J.1
-
25
-
-
33746466098
-
Mitochondrial mutations in cancer
-
Brandon, M., P. Baldi & D.C. Wallace. 2006. Mitochondrial mutations in cancer. Oncogene 25: 4647-4662.
-
(2006)
Oncogene
, vol.25
, pp. 4647-4662
-
-
Brandon, M.1
Baldi, P.2
Wallace, D.C.3
-
26
-
-
0034964421
-
Gene mutations in the succinate dehydrogenase subunit SDHB cause susceptibility to familial pheochromocytoma and to familial paraganglioma
-
Astuti, D. et al. 2001. Gene mutations in the succinate dehydrogenase subunit SDHB cause susceptibility to familial pheochromocytoma and to familial paraganglioma. Am. J. Hum. Genet . 69: 49-54.
-
(2001)
Am. J. Hum. Genet
, vol.69
, pp. 49-54
-
-
Astuti, D.1
-
27
-
-
0034602950
-
Mutations inSDHD, amitochondrial complex II gene, in hereditary paraganglioma
-
Baysal, B.E. et al. 2000.Mutations inSDHD, amitochondrial complex II gene, in hereditary paraganglioma. Science 287: 848-851.
-
(2000)
Science
, vol.287
, pp. 848-851
-
-
Baysal, B.E.1
-
28
-
-
28544446058
-
Mitochondrial tumour suppressors: A genetic and biochemical update
-
Gottlieb, E. & I.P. Tomlinson. 2005. Mitochondrial tumour suppressors: a genetic and biochemical update. Nat. Rev. Cancer. 5: 857-866.
-
(2005)
Nat. Rev. Cancer
, vol.5
, pp. 857-866
-
-
Gottlieb, E.1
Tomlinson, I.P.2
-
29
-
-
0033767445
-
Mutations in SDHC cause autosomal dominant paraganglioma, type 3
-
Niemann, S. & U. Muller. 2000. Mutations in SDHC cause autosomal dominant paraganglioma, type 3. Nat. Genet . 26: 268-270.
-
(2000)
Nat. Genet
, vol.26
, pp. 268-270
-
-
Niemann, S.1
Muller, U.2
-
30
-
-
19944433653
-
Succinate linksTCAcycle dysfunction to oncogenesis by inhibiting HIF-alpha prolyl hydroxylase
-
Selak, M.A. et al. 2005. Succinate linksTCAcycle dysfunction to oncogenesis by inhibiting HIF-alpha prolyl hydroxylase. Cancer Cell. 7: 77-85.
-
(2005)
Cancer Cell
, vol.7
, pp. 77-85
-
-
Selak, M.A.1
-
31
-
-
0034998621
-
Nearly all hereditary paragangliomas in the netherlands are caused by two founder mutations in the SDHD gene
-
Taschner, P.E. et al. 2001. Nearly all hereditary paragangliomas in the Netherlands are caused by two founder mutations in the SDHD gene. Genes Chromosomes Cancer. 31: 274-281.
-
(2001)
Genes Chromosomes Cancer
, vol.31
, pp. 274-281
-
-
Taschner, P.E.1
-
33
-
-
33746930794
-
Succinate dehydrogenase and fumarate hydratase: Linking mitochondrial dysfunction and cancer
-
King, A., M.A. Selak & E. Gottlieb. 2006. Succinate dehydrogenase and fumarate hydratase: linking mitochondrial dysfunction and cancer. Oncogene 25: 4675-4682.
-
(2006)
Oncogene
, vol.25
, pp. 4675-4682
-
-
King, A.1
Selak, M.A.2
Gottlieb, E.3
-
34
-
-
0024448458
-
Human cells lacking mtDNA: Repopulation with exogenousmitochondria by complementation
-
King,M.P. & G. Attardi. 1989. Human cells lacking mtDNA: repopulation with exogenousmitochondria by complementation. Science. 246: 500-503.
-
(1989)
Science
, vol.246
, pp. 500-503
-
-
King, M.P.1
Attardi, G.2
-
35
-
-
58149345071
-
MitochondrialDNAmutation stimulates prostate cancer growth in bone stromal environment
-
Arnold, R.S. et al. 2009.MitochondrialDNAmutation stimulates prostate cancer growth in bone stromal environment. Prostate. 69: 1-11.
-
(2009)
Prostate
, vol.69
, pp. 1-11
-
-
Arnold, R.S.1
-
36
-
-
33745700213
-
Defective oxidative phosphorylation in thyroid oncocytic carcinoma is associatedwith pathogenic mitochondrialDNAmutations affecting complexes i and III
-
Bonora, E. et al. 2006. Defective oxidative phosphorylation in thyroid oncocytic carcinoma is associatedwith pathogenic mitochondrialDNAmutations affecting complexes I and III. Cancer Res. 66: 6087-6096.
-
(2006)
Cancer Res
, vol.66
, pp. 6087-6096
-
-
Bonora, E.1
-
37
-
-
71749102014
-
Mitochondrial dysfunction in human breast cancer cells and their transmitochondrial cybrids
-
Ma, Y. et al. 2010. Mitochondrial dysfunction in human breast cancer cells and their transmitochondrial cybrids. Biochim. Biophys. Acta. 1797: 29-37.
-
(2010)
Biochim. Biophys. Acta.
, vol.1797
, pp. 29-37
-
-
Ma, Y.1
-
38
-
-
20044364344
-
Mt DNA mutations increase tumorigenicity in prostate cancer
-
Petros, J.A. et al. 2005. mtDNA mutations increase tumorigenicity in prostate cancer. Proc. Natl. Acad. Sci. USA 102: 719-724.
-
(2005)
Proc. Natl. Acad. Sci. USA
, vol.102
, pp. 719-724
-
-
Petros, J.A.1
-
39
-
-
16444386967
-
Positive contribution of pathogenic mutations in the mitochondrial genome to the promotion of cancer by prevention from apoptosis
-
Shidara, Y. et al. 2005. Positive contribution of pathogenic mutations in the mitochondrial genome to the promotion of cancer by prevention from apoptosis. Cancer Res. 65: 1655-1663.
-
(2005)
Cancer Res
, vol.65
, pp. 1655-1663
-
-
Shidara, Y.1
-
40
-
-
0033568447
-
Defective kinetics of cytochrome c oxidase and alteration of mitochondrial membrane potential in fibroblasts and cytoplasmic hybrid cells with the mutation for myoclonus epilepsy with ragged-red fibres ('MERRF') at position 8344 nt
-
Antonicka, H. et al. 1999. Defective kinetics of cytochrome c oxidase and alteration of mitochondrial membrane potential in fibroblasts and cytoplasmic hybrid cells with the mutation for myoclonus epilepsy with ragged-red fibres ('MERRF') at position 8344 nt. Biochem J . 342(Pt 3): 537-544.
-
(1999)
Biochem J
, vol.342
, Issue.PART 3
, pp. 537-544
-
-
Antonicka, H.1
-
41
-
-
0026608057
-
MELAS mutation in mtDNA binding site for transcription termination factor causes defects in protein synthesis and in respiration but no change in levels of upstream and downstreammature transcripts
-
Chomyn, A. et al. 1992. MELAS mutation in mtDNA binding site for transcription termination factor causes defects in protein synthesis and in respiration but no change in levels of upstream and downstreammature transcripts. Proc. Natl. Acad. Sci. USA 89: 4221-4225.
-
(1992)
Proc. Natl. Acad. Sci. USA
, vol.89
, pp. 4221-4225
-
-
Chomyn, A.1
-
42
-
-
0027936218
-
Cytoplasmic transfer of the mt DNA nt 8993 T→G (ATP6) pointmutation associated with Leigh syndrome into mtDNA-less cells demonstrates cosegregationwith a decrease in state III respiration and ADP/Oratio
-
Trounce, I., S. Neill & D.C. Wallace. 1994. Cytoplasmic transfer of the mtDNA nt 8993 T→G (ATP6) pointmutation associated with Leigh syndrome into mtDNA-less cells demonstrates cosegregationwith a decrease in state III respiration and ADP/Oratio. Proc.Natl. Acad. Sci.USA 91: 8334-8338.
-
(1994)
Proc.Natl. Acad. Sci.USA
, vol.91
, pp. 8334-8338
-
-
Trounce, I.1
Neill, S.2
Wallace, D.C.3
-
43
-
-
0037423202
-
Leber's hereditary optic neuropathy (LHON) pathogenic mutations induce mitochondrialdependent apoptotic death in transmitochondrial cells incubatedwith galactosemedium
-
Ghelli, A. et al. 2003. Leber's hereditary optic neuropathy (LHON) pathogenic mutations induce mitochondrialdependent apoptotic death in transmitochondrial cells incubatedwith galactosemedium. J. Biol.Chem. 278: 4145-4150.
-
(2003)
J. Biol.Chem
, vol.278
, pp. 4145-4150
-
-
Ghelli, A.1
-
44
-
-
53049084911
-
Enhanced glycolysis induced by mt DNA mutations does not regulate metastasis
-
Ishikawa, K. et al. 2008. Enhanced glycolysis induced by mtDNA mutations does not regulate metastasis. FEBS Lett . 582: 3525-3530.
-
(2008)
FEBS Lett
, vol.582
, pp. 3525-3530
-
-
Ishikawa, K.1
-
45
-
-
66749150948
-
How do human cells react to the absence of mitochondrial DNA?
-
Mineri, R. et al. 2009. How do human cells react to the absence of mitochondrial DNA? PLoS One. 4: e5713.
-
(2009)
PLoS One
, vol.4
-
-
Mineri, R.1
-
46
-
-
58149299283
-
Mt DNA depletion confers specific gene expression profiles in human cells grown in culture and in xenograft
-
Magda, D. et al. 2008. mtDNA depletion confers specific gene expression profiles in human cells grown in culture and in xenograft. BMC Genom. 9: 521.
-
(2008)
BMC Genom
, vol.9
, pp. 521
-
-
Magda, D.1
-
47
-
-
0026697079
-
Recovery of themissing tumorigenicity inmitochondrialDNA-lessHeLa cells by introduction of mitochondrial DNA from normal human cells
-
Hayashi, J., M. Takemitsu & I. Nonaka. 1992. Recovery of themissing tumorigenicity inmitochondrialDNA-lessHeLa cells by introduction of mitochondrial DNA from normal human cells. Somat. Cell Mol. Genet . 18: 123-129.
-
(1992)
Somat. Cell Mol. Genet
, vol.18
, pp. 123-129
-
-
Hayashi, J.1
Takemitsu, M.2
Nonaka, I.3
-
48
-
-
0030728737
-
Diminished tumorigenic phenotype after depletion of mitochondrial DNA
-
Cavalli, L.R., M. Varella-Garcia & B.C. Liang. 1997. Diminished tumorigenic phenotype after depletion of mitochondrial DNA. Cell Growth Differ. 8: 1189-1198.
-
(1997)
Cell Growth Differ
, vol.8
, pp. 1189-1198
-
-
Cavalli, L.R.1
Varella-Garcia, M.2
Liang, B.C.3
-
49
-
-
22044444055
-
Enhancement of cisplatin-induced apoptosis and caspase 3 activation by depletion of mitochondrial DNA in a human osteosarcoma cell line
-
Yen, H.C. et al. 2005. Enhancement of cisplatin-induced apoptosis and caspase 3 activation by depletion of mitochondrial DNA in a human osteosarcoma cell line. Ann. N.Y. Acad Sci. 1042: 516-522.
-
(2005)
Ann. N.Y. Acad Sci
, vol.1042
, pp. 516-522
-
-
Yen, H.C.1
-
50
-
-
48849088703
-
Impact of mitochondrial DNA on hypoxic radiation sensitivity in human fibroblast cells and osteosarcoma cell lines
-
Yamazaki, H. et al. 2008. Impact of mitochondrial DNA on hypoxic radiation sensitivity in human fibroblast cells and osteosarcoma cell lines. Oncol Rep. 19: 1545-1549.
-
(2008)
Oncol Rep
, vol.19
, pp. 1545-1549
-
-
Yamazaki, H.1
-
51
-
-
0037038669
-
Mitochondrial stress-induced calcium signaling, phenotypic changes and invasive behavior in human lung carcinoma A549 cells
-
Amuthan, G. et al. 2002. Mitochondrial stress-induced calcium signaling, phenotypic changes and invasive behavior in human lung carcinoma A549 cells. Oncogene. 21: 7839-7849.
-
(2002)
Oncogene
, vol.21
, pp. 7839-7849
-
-
Amuthan, G.1
-
52
-
-
77954586489
-
Inhibition of mitochondrial respiration mediates apoptosis induced by the anti-tumoral alkaloid lamellarin D
-
Ballot,C. et al. 2010. Inhibition of mitochondrial respiration mediates apoptosis induced by the anti-tumoral alkaloid lamellarin D. Apoptosis [Epub ahead of print].
-
Apoptosis [Epub Ahead of Print]
, vol.2010
-
-
Ballot, C.1
-
53
-
-
56449097919
-
Tumorigenic transformation of humanbreast epithelial cells induced by mitochondrial DNA depletion
-
Kulawiec, M. et al. 2008. Tumorigenic transformation of humanbreast epithelial cells induced by mitochondrialDNA depletion. Cancer Biol Ther. 7: 1732-1743.
-
(2008)
Cancer Biol Ther
, vol.7
, pp. 1732-1743
-
-
Kulawiec, M.1
-
54
-
-
39649084663
-
Induction of acquired resistance to antiestrogen by reversible mitochondrial DNA depletion in breast cancer cell line
-
Naito, A. et al. 2008. Induction of acquired resistance to antiestrogen by reversible mitochondrial DNA depletion in breast cancer cell line. Int J Cancer. 122: 1506-1511.
-
(2008)
Int J Cancer
, vol.122
, pp. 1506-1511
-
-
Naito, A.1
-
55
-
-
23644434087
-
Inter-genomic cross talk between mitochondria and the nucleus plays an important role in tumorigenesis
-
Singh, K.K. et al. 2005. Inter-genomic cross talk between mitochondria and the nucleus plays an important role in tumorigenesis. Gene. 354: 140-146.
-
(2005)
Gene
, vol.354
, pp. 140-146
-
-
Singh, K.K.1
-
56
-
-
59149087660
-
Nicotine induces resistance to chemotherapy by modulating mitochondrial signaling in lung cancer
-
Zhang, J. et al. 2009. Nicotine induces resistance to chemotherapy by modulating mitochondrial signaling in lung cancer. Am J Respir Cell Mol Biol. 40: 135-146.
-
(2009)
Am J Respir Cell Mol Biol
, vol.40
, pp. 135-146
-
-
Zhang, J.1
-
57
-
-
1442340729
-
Nuclear genes involved in mitochondria-to-nucleus communication in breast cancer cells
-
Delsite, R. et al. 2002. Nuclear genes involved in mitochondria-to- nucleus communication in breast cancer cells. Mol Cancer. 1: 6.
-
(2002)
Mol Cancer
, vol.1
, pp. 6
-
-
Delsite, R.1
-
58
-
-
34250355048
-
Mitochondrial genetic background modifies breast cancer risk
-
Bai, R.K. et al. 2007. Mitochondrial genetic background modifies breast cancer risk. Cancer Res. 67: 4687-4694.
-
(2007)
Cancer Res
, vol.67
, pp. 4687-4694
-
-
Bai, R.K.1
-
59
-
-
52649175410
-
Mitochondrial DNA variant interactions modify breast cancer risk
-
Covarrubias, D. et al. 2008. Mitochondrial DNA variant interactions modify breast cancer risk. J. Hum. Genet . 53: 924-928.
-
(2008)
J. Hum. Genet
, vol.53
, pp. 924-928
-
-
Covarrubias, D.1
-
60
-
-
24744442376
-
Mitochondrial DNA G10398A polymorphism and invasive breast cancer in African-American women
-
Canter, J.A. et al. 2005.Mitochondrial DNA G10398A polymorphism and invasive breast cancer in African-American women. Cancer Res. 65: 8028-8033.
-
(2005)
Cancer Res
, vol.65
, pp. 8028-8033
-
-
Canter, J.A.1
-
61
-
-
33947373403
-
MitochondrialDNA G10398A polymorphism impartsmaternal Haplogroup N a risk for breast and esophageal cancer
-
Darvishi, K. et al. 2007.MitochondrialDNA G10398A polymorphism impartsmaternal Haplogroup N a risk for breast and esophageal cancer. Cancer Lett . 249: 249-255.
-
(2007)
Cancer Lett
, vol.249
, pp. 249-255
-
-
Darvishi, K.1
-
62
-
-
74049108985
-
Mt DNA G10398A variant in African-American women with breast cancer provides resistance to apoptosis and promotes metastasis in mice
-
Kulawiec, M., K.M. Owens & K.K. Singh. 2009. mtDNA G10398A variant in African-American women with breast cancer provides resistance to apoptosis and promotes metastasis in mice. J. Hum. Genet . 54: 647-654.
-
(2009)
J. Hum. Genet
, vol.54
, pp. 647-654
-
-
Kulawiec, M.1
Owens, K.M.2
Singh, K.K.3
-
63
-
-
49749150948
-
Progressive tumor features accompany epithelial-mesenchymal transition induced in mitochondrial DNA-depleted cells
-
Naito, A. et al. 2008. Progressive tumor features accompany epithelial-mesenchymal transition induced in mitochondrial DNA-depleted cells. Cancer Sci. 99: 1584-1588.
-
(2008)
Cancer Sci
, vol.99
, pp. 1584-1588
-
-
Naito, A.1
|