Accumulation of oxidative DNA damage in brain mitochondria in mouse model of hereditary ferritinopathy

Neuroscience Letters

Volumn 479, Issue 1, 2010, Pages 44-48

  Deng, Xiaoling ^a   Vidal, Ruben ^b   Englander, Ella W ^a  

^a University of Texas Medical Branch School of Medicine   (United States)

^b INDIANA UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Brain; DNA damage; Ferritin; Hereditary ferritinopathy; Iron overload; Mitochondria

Indexed keywords

FERRITIN; GUANINE; MITOCHONDRIAL DNA; APOFERRITIN; DNA ADDUCT; IRON;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BIOACCUMULATION; BRAIN LEVEL; BRAIN MITOCHONDRION; CONTROLLED STUDY; DISEASE COURSE; DNA ADDUCT; DNA DAMAGE; DNA MODIFICATION; HEMOSIDEROSIS; HEREDITARY FERRITINOPATHY; IRON METABOLISM; IRON OVERLOAD; LIGHT CHAIN; MOUSE; NERVE CELL NECROSIS; NERVE DEGENERATION; NEUROPATHOLOGY; NEUROTOXICITY; NONHUMAN; OXIDATIVE STRESS; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; TRANSGENIC MOUSE; AGING; ANIMAL; BRAIN; C57BL MOUSE; CELL NUCLEUS; DISEASE MODEL; DISEASE PREDISPOSITION; GENETICS; METABOLIC ENCEPHALOPATHY; METABOLISM; MITOCHONDRION; PATHOLOGY;

AGING; ANIMALS; APOFERRITINS; BRAIN; BRAIN DISEASES, METABOLIC; CELL NUCLEUS; DISEASE MODELS, ANIMAL; DISEASE SUSCEPTIBILITY; DNA ADDUCTS; DNA DAMAGE; DNA, MITOCHONDRIAL; FERRITINS; IRON; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MITOCHONDRIA; OXIDATIVE STRESS;

EID: 77953713610     PISSN: 03043940     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neulet.2010.05.025     Document Type: Article

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