The role of TLR2, TRL3, TRL4, and TRL9 signaling in the pathogenesis of autoimmune disease in a retinal autoimmunity model

Investigative Ophthalmology and Visual Science

Volumn 51, Issue 6, 2010, Pages 3092-3099

  Fang, Jiazhu ^a   Silver, Phyllis B ^b   Fang, Dan ^a   Wen, Feng ^a   Li, Bing ^a   Ren, Xiangrong ^a   Caspi, Rachel R ^b   Su, Shao Bo ^a  

^a SUN YAT SEN UNIVERSITY   (China)

^b NATIONAL EYE INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GAMMA INTERFERON; INTERLEUKIN 17; LIPOPOLYSACCHARIDE; POLYMYXIN B; TOLL LIKE RECEPTOR 2; TOLL LIKE RECEPTOR 3; TOLL LIKE RECEPTOR 4; TOLL LIKE RECEPTOR 9; TOLL LIKE RECEPTOR AGONIST; AUTOANTIGEN; EYE PROTEIN; INTERPHOTORECEPTOR RETINOID BINDING PROTEIN; INTERSTITIAL RETINOL-BINDING PROTEIN; MYELOID DIFFERENTIATION FACTOR 88; RETINOL BINDING PROTEIN; TLR2 PROTEIN, MOUSE; TLR3 PROTEIN, MOUSE; TLR4 PROTEIN, MOUSE; TLR9 PROTEIN, MOUSE; TOLL LIKE RECEPTOR;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOIMMUNE DISEASE; AUTOIMMUNE UVEITIS; CONTROLLED STUDY; DELAYED HYPERSENSITIVITY; FEMALE; IMMUNIZATION; LYMPHOCYTE PROLIFERATION; MALE; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; SCORING SYSTEM; SIGNAL TRANSDUCTION; T LYMPHOCYTE; UVEITIS; ANIMAL; AUTOIMMUNITY; C57BL MOUSE; DISEASE MODEL; DRUG POTENTIATION; GENE SILENCING; IMMUNOLOGY; LYMPHOCYTE ACTIVATION; METABOLISM; MOUSE MUTANT; PHYSIOLOGY;

ANIMALS; AUTOANTIGENS; AUTOIMMUNE DISEASES; AUTOIMMUNITY; DISEASE MODELS, ANIMAL; EYE PROTEINS; FEMALE; GENE SILENCING; HYPERSENSITIVITY, DELAYED; INTERFERON-GAMMA; INTERLEUKIN-17; LIPOPOLYSACCHARIDES; LYMPHOCYTE ACTIVATION; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYELOID DIFFERENTIATION FACTOR 88; RETINOL-BINDING PROTEINS; SIGNAL TRANSDUCTION; T-LYMPHOCYTES; TOLL-LIKE RECEPTOR 2; TOLL-LIKE RECEPTOR 3; TOLL-LIKE RECEPTOR 4; TOLL-LIKE RECEPTOR 9; TOLL-LIKE RECEPTORS; UVEITIS;

EID: 77953246527     PISSN: 01460404     EISSN: 15525783     Source Type: Journal    
DOI: 10.1167/iovs.09-4754     Document Type: Article

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