Externally triggered egress is the major fate of Toxoplasma gondii during acute infection

Journal of Immunology

Volumn 183, Issue 10, 2009, Pages 6667-6680

  Tomita, Tadakimi ^a   Yamada, Tatsuya ^a   Weiss, Louis M ^a   Orlofsky, Amos ^a  

^a ALBERT EINSTEIN COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

4 (4 FLUOROPHENYL) 2 (4 METHYLSULFINYLPHENYL) 5 (4 PYRIDYL)IMIDAZOLE; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; MITOGEN ACTIVATED PROTEIN KINASE P38; NITROGEN DERIVATIVE; REACTIVE OXYGEN METABOLITE;

ADOPTIVE TRANSFER; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CALCIUM CELL LEVEL; CELL CULTURE; CELL DIVISION; CONTROLLED STUDY; ENZYME INHIBITION; HOST PARASITE INTERACTION; HOST RESISTANCE; INFECTION; INFLAMMATORY CELL; MACROPHAGE; MOUSE; NONHUMAN; PRIORITY JOURNAL; REGULATORY MECHANISM; REINFECTION; SIGNAL TRANSDUCTION; TARGET CELL; TOXOPLASMA GONDII; TOXOPLASMOSIS;

ACUTE DISEASE; ADOPTIVE TRANSFER; ANIMALS; CALCIUM; ENZYME INHIBITORS; HOST-PARASITE INTERACTIONS; MACROPHAGES; MICE; MICE, INBRED C57BL; PROTEIN KINASE INHIBITORS; PROTEIN KINASES; TOXOPLASMA; TOXOPLASMOSIS;

EID: 77951930485     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.0900516     Document Type: Article

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