Hes1 expression is reduced in Tbx1 null cells and is required for the development of structures affected in 22q11 deletion syndrome

Developmental Biology

Volumn 340, Issue 2, 2010, Pages 369-380

  van Bueren, Kelly Lammerts ^a   Papangeli, Irinna ^a   Rochais, Francesca ^b   Pearce, Kerra ^a   Roberts, Catherine ^a   Calmont, Amelie ^a   Szumska, Dorota ^c   Kelly, Robert G ^b   Bhattacharya, Shoumo ^c   Scambler, Peter J ^a  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

^b AIX MARSEILLE UNIVERSITÉ   (France)

^c UNIVERSITY OF OXFORD   (United Kingdom)

Author keywords

22q11; FACS; Hes1; Microarray; Mouse; Pharyngeal arch artery; Tbx1; Thymus

Indexed keywords

BETA GALACTOSIDASE; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR HES 1; TRANSCRIPTION FACTOR TBX1; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CELL ISOLATION; CHROMOSOME DELETION; CHROMOSOME DELETION 22; CONGENITAL HEART MALFORMATION; CONTROLLED STUDY; CRANIOFACIAL MALFORMATION; EMBRYO; FLUORESCENCE ACTIVATED CELL SORTING; GENE EXPRESSION; GENE EXPRESSION PROFILING; HETEROZYGOTE; MALFORMATION SYNDROME; MOUSE; MUTAGENESIS; NONHUMAN; NULL CELL; PHARYNX DISEASE; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; THYMUS DISEASE; TRANSCRIPTION REGULATION;

MUS;

EID: 77951203831     PISSN: 00121606     EISSN: 1095564X     Source Type: Journal    
DOI: 10.1016/j.ydbio.2010.01.020     Document Type: Article

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