NSAID-activated gene-1 as a molecular target for capsaicin-induced apoptosis through a novel molecular mechanism involving GSK3β, C/EBPβ and ATF3

Carcinogenesis

Volumn 31, Issue 4, 2010, Pages 719-728

  Lee, Seong Ho ^a   Krisanapun, Chutwadee ^b   Baek, Seung Joon ^a  

^a UNIVERSITY OF TENNESSEE   (United States)

^b SRINAKHARINWIROT UNIVERSITY   (Thailand)

Author keywords

[No Author keywords available]

Indexed keywords

ACTIVATING TRANSCRIPTION FACTOR 3; CAPSAICIN; CCAAT ENHANCER BINDING PROTEIN BETA; GLYCOGEN SYNTHASE KINASE 3BETA; NONSTEROID ANTIINFLAMMATORY DRUG ACTIVATED GENE 1 PROTEIN; PROTEIN KINASE C; UNCLASSIFIED DRUG; ATF3 PROTEIN, HUMAN; GDF15 PROTEIN, HUMAN; GLYCOGEN SYNTHASE KINASE 3; GLYCOGEN SYNTHASE KINASE 3 BETA; GROWTH DIFFERENTIATION FACTOR 15; RETINOIC ACID RECEPTOR; RETINOIC ACID RECEPTOR ALPHA;

ANTINEOPLASTIC ACTIVITY; APOPTOSIS; ARTICLE; BINDING SITE; CANCER CELL CULTURE; CANCER INHIBITION; COLORECTAL CANCER; CONTROLLED STUDY; DRUG TARGETING; GENE EXPRESSION REGULATION; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN PHOSPHORYLATION; PROTEIN PROTEIN INTERACTION; SIGNAL TRANSDUCTION; TRANSACTIVATION; UPREGULATION; CELL STRAIN HCT116; CELL STRAIN HT29; COLORECTAL TUMOR; DRUG EFFECT; GENETICS; PATHOLOGY; PHOSPHORYLATION; PHYSIOLOGY;

ACTIVATING TRANSCRIPTION FACTOR 3; APOPTOSIS; CAPSAICIN; CCAAT-ENHANCER-BINDING PROTEIN-BETA; COLORECTAL NEOPLASMS; GLYCOGEN SYNTHASE KINASE 3; GROWTH DIFFERENTIATION FACTOR 15; HCT116 CELLS; HT29 CELLS; HUMANS; PHOSPHORYLATION; PROMOTER REGIONS, GENETIC; PROTEIN KINASE C; RECEPTORS, RETINOIC ACID; SIGNAL TRANSDUCTION;

EID: 77950896712     PISSN: 01433334     EISSN: 14602180     Source Type: Journal    
DOI: 10.1093/carcin/bgq016     Document Type: Article

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