The suppression of MAD1 by AKT-mediated phosphorylation activates MAD1 target genes transcription

Molecular Carcinogenesis

Volumn 48, Issue 11, 2009, Pages 1048-1058

  Chou, Chao Kai ^a,b   Lee, Dung Fang ^a,b   Sun, Hui Lung ^a   Li, Long Yuan ^c,d   Lin, Chun Yi ^c   Huang, Wei Chien ^a,c,d   Hsu, Jung Mao ^a,b   Kuo, Hsu Ping ^a,b   Yamaguchi, Hirohito ^a   Wang, Ying Nai ^a   Mo, Liu ^a,b   Wu, Hsin Yi ^e   Liao, Pao Chi ^e   Yen, Chia Jui ^a   Hung, Mien Chie ^a,b,c,d  

^a UNIVERSITY OF TEXAS MD ANDERSON CANCER CENTER   (United States)

^b UNIVERSITY OF TEXAS   (United States)

^c CHINA MEDICAL UNIVERSITY   (Taiwan)

^d ASIA UNIVERSITY   (Taiwan)

^e NATIONAL CHENG KUNG UNIVERSITY   (Taiwan)

Author keywords

AKT; hTERT; MAD1

Indexed keywords

ALANINE; PROTEIN KINASE B; PROTEIN MAD1; SERINE;

AMINO ACID SUBSTITUTION; ARTICLE; BINDING AFFINITY; CELL CYCLE REGULATION; CELL GROWTH; CONTROLLED STUDY; DNA BINDING; ENZYME INHIBITION; GENE EXPRESSION; GENE TARGETING; HUMAN; HUMAN CELL; ONCOGENE MYC; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN DNA BINDING; PROTEIN PHOSPHORYLATION; PROTEIN PROTEIN INTERACTION; TRANSCRIPTION REGULATION;

CELL CYCLE PROTEINS; CELL LINE, TUMOR; CHROMATIN IMMUNOPRECIPITATION; GENE EXPRESSION REGULATION; GENES, MYC; HUMANS; MASS SPECTROMETRY; NUCLEAR PROTEINS; PHOSPHORYLATION; PROTO-ONCOGENE PROTEINS C-AKT; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; TRANSCRIPTION, GENETIC;

EID: 70350464148     PISSN: 08991987     EISSN: 10982744     Source Type: Journal    
DOI: 10.1002/mc.20557     Document Type: Article

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