Impact of Sur1 gene inactivation on the morphology of mouse pancreatic endocrine tissue

Cell and Tissue Research

Volumn 335, Issue 3, 2009, Pages 505-515

  Marhfour, Ihsane ^a   Moulin, Pierre ^a   Marchandise, Joëlle ^a   Rahier, Jacques ^a   Sempoux, Christine ^a   Guiot, Yves ^a,b  

^a UNIVERSITÉ CATHOLIQUE DE LOUVAIN   (Belgium)

^b CLINIQUES UNIVERSITAIRES SAINT LUC   (Belgium)

Author keywords

Congenital hyperinsulinism; Endocrine pancreatic cells; K ATP channel; Morphology; Sur1 knockout mice

Indexed keywords

GLUCAGON; GLUCOSE TRANSPORTER; INSULIN; MESSENGER RNA; SULFONYLUREA RECEPTOR 1; ABC TRANSPORTER; DRUG RECEPTOR; INWARDLY RECTIFYING POTASSIUM CHANNEL; SULFONYLUREA RECEPTOR;

ADAPTIVE IMMUNITY; ADULT; AGE DISTRIBUTION; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CELL FUNCTION; CELL INTERACTION; CELL SIZE; CELL STRUCTURE; CELLULAR DISTRIBUTION; CONTROLLED STUDY; FEMALE; GENE EXPRESSION; GENE INACTIVATION; GLUT 2 GENE; IMMUNOHISTOCHEMISTRY; INSULIN RELEASE; INSULINEMIA; JUVENILE ANIMAL; MORPHOMETRY; MOUSE; NONHUMAN; PANCREAS ISLET ALPHA CELL; PANCREAS ISLET BETA CELL; PANCREAS TISSUE; PERSISTENT HYPERINSULINEMIC HYPOGLYCEMIA OF INFANCY; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN MODIFICATION; SUR1 GENE; WILD TYPE MOUSE; AGE; ANIMAL; BLOOD; C57BL MOUSE; CYTOLOGY; GENE SILENCING; GENETICS; GLUCOSE BLOOD LEVEL; HISTOLOGY; METABOLISM; MOUSE MUTANT; PANCREAS; PANCREAS ISLET; PHYSIOLOGY; SECRETION;

AGE FACTORS; ANIMALS; ATP-BINDING CASSETTE TRANSPORTERS; BLOOD GLUCOSE; FEMALE; GENE SILENCING; GLUCAGON; IMMUNOHISTOCHEMISTRY; INSULIN; ISLETS OF LANGERHANS; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; PANCREAS; POTASSIUM CHANNELS, INWARDLY RECTIFYING; RECEPTORS, DRUG;

EID: 67651151568     PISSN: 0302766X     EISSN: 14320878     Source Type: Journal    
DOI: 10.1007/s00441-008-0733-2     Document Type: Article

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