STAT3 deletion sensitizes cells to oxidative stress

Biochemical and Biophysical Research Communications

Volumn 385, Issue 3, 2009, Pages 324-329

  Barry, Seán P ^a   Townsend, Paul A ^b   McCormick, James ^a   Knight, Richard A ^a   Scarabelli, Tiziano M ^c   Latchman, David S ^a   Stephanou, Anastasis ^a  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

^b UNIVERSITY OF SOUTHAMPTON   (United Kingdom)

^c ST JOHN HOSPITAL AND MEDICAL CENTER   (United States)

Author keywords

Apoptosis; Ischemia; Myocardial infarction; Oxidative stress; STAT1; STAT3

Indexed keywords

JANUS KINASE; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B; STAT3 PROTEIN;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL DEATH; CELL PROTECTION; COMPARATIVE STUDY; CONTROLLED STUDY; HEART MUSCLE CELL; ISCHEMIA; NEWBORN; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; REPERFUSION INJURY;

ANIMALS; APOPTOSIS; CELL LINE; GENE DELETION; MICE; MUTATION; MYOCARDIAL REPERFUSION INJURY; MYOCYTES, CARDIAC; OXIDATIVE STRESS; RATS; STAT3 TRANSCRIPTION FACTOR;

ADENOVIRIDAE; RATTUS;

EID: 67349242289     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2009.05.051     Document Type: Article

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