Diseases that may benefit from manipulating the Th17 pathway

European Journal of Immunology

Volumn 39, Issue 3, 2009, Pages 667-669

  Miossec, Pierre ^a  

^a University of Lyon   (France)

Author keywords

Destruction; Diseases; IL 17; Inflammation; Th17

Indexed keywords

INTERLEUKIN 1; INTERLEUKIN 16 INHIBITOR; INTERLEUKIN 17; INTERLEUKIN 17 INHIBITOR; INTERLEUKIN 21; INTERLEUKIN 22; INTERLEUKIN 6; TUMOR NECROSIS FACTOR; TUMOR NECROSIS FACTOR INHIBITOR; UNCLASSIFIED DRUG; CYTOKINE;

ARTICLE; BACTERIAL INFECTION; BACTERIAL PERITONITIS; BONE DESTRUCTION; BONE MARROW; CELL INTERACTION; CLINICAL TRIAL; CROHN DISEASE; CYTOKINE PRODUCTION; DISEASE SEVERITY; HUMAN; IMMUNE RESPONSE; IMMUNOPATHOGENESIS; INFLAMMATION; LUNG INFECTION; LYMPHOCYTE MIGRATION; MULTIPLE SCLEROSIS; NEUTROPHIL; NONHUMAN; OSTEOPOROSIS; PRIORITY JOURNAL; PROTEIN TARGETING; PSORIASIS; REGULATORY T LYMPHOCYTE; RHEUMATOID ARTHRITIS; SIGNAL TRANSDUCTION; T LYMPHOCYTE ACTIVATION; T LYMPHOCYTE SUBPOPULATION; TH17 CELL; ACUTE DISEASE; ANIMAL; AUTOIMMUNE DISEASE; CHRONIC DISEASE; DRUG ANTAGONISM; HELPER CELL; IMMUNOLOGY; INFECTION; METABOLISM; MOUSE;

ACUTE DISEASE; ANIMALS; AUTOIMMUNE DISEASES; CHRONIC DISEASE; CYTOKINES; HUMANS; INFECTION; INFLAMMATION; INTERLEUKIN-17; MICE; T-LYMPHOCYTES, HELPER-INDUCER;

EID: 64049094332     PISSN: 00142980     EISSN: 15214141     Source Type: Journal    
DOI: 10.1002/eji.200839088     Document Type: Article

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