Senescence accelerated mouse strain is sensitive to neurodegeneration induced by mild impairment of oxidative metabolism

Brain Research

Volumn 1264, Issue , 2009, Pages 111-118

  Zhang, Qipeng ^a   Ding, Hanqing ^b   Li, Wenxia ^b   Fan, Zhiqin ^b   Sun, Anyang ^a   Luo, Jia ^b,c   Ke, Zun Ji ^b  

^a FUDAN UNIVERSITY   (China)

^b Shanghai Jiao Tong University Affiliated Sixth People s Hospital   (China)

^c UNIVERSITY OF KENTUCKY   (United States)

Author keywords

APP; Neuronal death; Oxidative stress; Senescence accelerated mice; Thiamine deficiency

Indexed keywords

AMYLOID PRECURSOR PROTEIN;

AEROBIC METABOLISM; AGING; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; IMMUNOREACTIVITY; MALE; MOUSE; NERVE DEGENERATION; NEUROPATHOLOGY; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; SENESCENCE; SENESCENCE ACCELERATED MOUSE; STRAIN DIFFERENCE; THALAMUS NUCLEUS; THIAMINE DEFICIENCY; WEIGHT REDUCTION;

AMYLOID BETA-PROTEIN PRECURSOR; ANALYSIS OF VARIANCE; ANIMALS; CELL COUNT; IMMUNOHISTOCHEMISTRY; MALE; MICE; MICE, INBRED C57BL; MICE, MUTANT STRAINS; NERVE TISSUE PROTEINS; NEURODEGENERATIVE DISEASES; NEURONS; NUCLEAR PROTEINS; OXIDATIVE STRESS; PYRIDINIUM COMPOUNDS; THALAMUS; THIAMINE DEFICIENCY; WEIGHT LOSS;

ANIMALIA; MUS;

EID: 63249118896     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.brainres.2009.02.005     Document Type: Article

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