The transition of closely opposed lesions to double-strand breaks during long-patch base excision repair is prevented by the coordinated action of DNA polymerase δ and Rad27/Fen1

Molecular and Cellular Biology

Volumn 29, Issue 5, 2009, Pages 1212-1221

  Ma, Wenjian ^a   Panduri, Vijayalakshmi ^a   Sterling, Joan F ^a   Van Houten, Bennett ^a,b   Gordenin, Dmitry A ^a   Resnick, Michael A ^a  

^a NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES   (United States)

^b UNIVERSITY OF PITTSBURGH CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

COMPLEMENTARY DNA; DNA DIRECTED DNA POLYMERASE DELTA; ENDONUCLEASE; MESYLIC ACID METHYL ESTER; RAD27 PROTEIN; UNCLASSIFIED DRUG; DNA DIRECTED DNA POLYMERASE GAMMA; FLAP ENDONUCLEASE; RAD27 PROTEIN, S CEREVISIAE; SACCHAROMYCES CEREVISIAE PROTEIN;

ALKYLATION; ARTICLE; DNA DAMAGE; DNA REPAIR; DOUBLE STRANDED DNA BREAK; EXCISION REPAIR; FUNGAL STRAIN; NONHUMAN; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; PROTEIN FUNCTION; SACCHAROMYCES CEREVISIAE; YEAST CELL; GENETICS; MUTATION; PHYSIOLOGY;

DNA BREAKS, DOUBLE-STRANDED; DNA POLYMERASE III; DNA REPAIR; FLAP ENDONUCLEASES; METHYL METHANESULFONATE; MUTATION; POLYMERASE CHAIN REACTION; SACCHAROMYCES CEREVISIAE; SACCHAROMYCES CEREVISIAE PROTEINS;

SACCHAROMYCES CEREVISIAE; SACCHAROMYCETALES;

EID: 61749094513     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.01499-08     Document Type: Article

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