Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-d-aspartate receptors

Neuroscience

Volumn 158, Issue 1, 2009, Pages 344-352

  Wahl, A S ^a   Buchthal, B ^a   Rode, F ^b   Bomholt, S F ^b   Freitag, H E ^a   Hardingham, G E ^c   Ronn L C B ^b   Bading, H ^a  

^a UNIVERSITY OF HEIDELBERG   (Germany)

^b NEUROSEARCH A S   (Denmark)

^c UNIVERSITY OF EDINBURGH   (United Kingdom)

Author keywords

Clca1; gene expression; marker; NMDA receptor; stroke; transient ischemia

Indexed keywords

CALCIUM ACTIVATED CHLORIDE CHANNEL; CHLORIDE CHANNEL; GLUCOSE; MESSENGER RNA; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; OXYGEN; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN HYPOXIA; BRAIN ISCHEMIA; CAROTID ARTERY OBSTRUCTION; CELL SURVIVAL; CLCA1 GENE; CONTROLLED STUDY; DISEASE MARKER; FOREBRAIN; GENE; GENE ACTIVATION; GENE CONTROL; GENE EXPRESSION; GENE INDUCTION; GENETIC TRANSCRIPTION; GENOME; GERBIL; HIPPOCAMPUS; MALE; MOUSE; NERVE CELL LESION; NEWBORN; NONHUMAN; ONCOGENE C FOS; PRIORITY JOURNAL; RAT; RECEPTOR UPREGULATION; SIGNAL TRANSDUCTION;

ACTION POTENTIALS; ANIMALS; BIOLOGICAL MARKERS; CELLS, CULTURED; CHLORIDE CHANNELS; GENE EXPRESSION REGULATION; GERBILLINAE; HIPPOCAMPUS; HYPOXIA-ISCHEMIA, BRAIN; MALE; MICE; MICE, INBRED C57BL; NERVE DEGENERATION; NERVE NET; NEURONS; PROTO-ONCOGENE PROTEINS C-FOS; RECEPTORS, N-METHYL-D-ASPARTATE; RNA, MESSENGER; SYNAPSES; SYNAPTIC TRANSMISSION;

EID: 58149477890     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2008.06.018     Document Type: Article

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