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1
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40049097821
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K/DOQl-recommended intact PTH • levels do not prevent low-turnover bone disease in hemodialysis patients
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This histologic study in one dialysis center shows that implementation of the K/DOQl recommendation on PTH levels did not prevent ABD
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Barreto FC, Barreto DV, Moyses RM, et al. K/DOQl-recommended intact PTH • levels do not prevent low-turnover bone disease in hemodialysis patients. Kidney Int 2008; 73:771 -777. This histologic study in one dialysis center shows that implementation of the K/DOQl recommendation on PTH levels did not prevent ABD.
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Barreto, F.C.1
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K/DOQI clinical practice guidelines for management of dyslipidemias in patients with kidney disease. Am J Kidney Dis 2003; 41 :I-IV, S1-S91.
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The lack of accuracy of presently available PTH assays and the limitation caused to the management of ROD are reviewed
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Martin KJ, Gonzalez EA. Parathyroid hormone assay: problems and oppor- •• tunities. Pediatr Nephrol 2007; 22:1651-1654. The lack of accuracy of presently available PTH assays and the limitation caused to the management of ROD are reviewed.
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48049102175
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The mechanism of phosphorus as a •• cardiovascular risk factor in CKD
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This in-vitro study explores the mechanism of vascular calcification and demonstrates that hyperphosphatemia has an active role, by inducing osterix, in addition to contributing to a high Ca x P product
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Mathew S, Tustison KS, Sugatani T, et al. The mechanism of phosphorus as a •• cardiovascular risk factor in CKD. J Am Soc Nephrol 2008; 19:1092 -1105. This in-vitro study explores the mechanism of vascular calcification and demonstrates that hyperphosphatemia has an active role, by inducing osterix, in addition to contributing to a high Ca x P product.
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J Am Soc Nephrol
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Mathew, S.1
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Hyperphosphatemia of chronic kidney disease
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Hruska KA, Mathew S, Lund R, et al. Hyperphosphatemia of chronic kidney disease. Kidney Int 2008; 74:148-157.
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Hruska, K.A.1
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40949158527
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BMP-2 promotes phosphate uptake, phenotypic modulation, and calcification of human vascular smooth muscle cells
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London GM, Marchais SJ, Guerin AP, et al. Association of bone activity, •• calcium load, aortic stiffness, and calcifications in ESRD. J Am Soc Nephrol 2008; 19:1827-1835. This important study shows that the amount of aortic calcification associated with oral calcium load is a function of bone turnover and is increased when bone turnover is decreased.
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London GM, Marchais SJ, Guerin AP, et al. Association of bone activity, •• calcium load, aortic stiffness, and calcifications in ESRD. J Am Soc Nephrol 2008; 19:1827-1835. This important study shows that the amount of aortic calcification associated with oral calcium load is a function of bone turnover and is increased when bone turnover is decreased.
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15
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34548274602
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Differential effects of vitamin D analogs on vascular calcification
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Vitamin D receptor activators can • protect against vascular calcification
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The effect of vitamin D receptor activators on vascular calcification depends on their dosage. Low doses can be protective by inhibiting PTH secretion
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Mathew S, Lund RJ, Chaudhary LR, et al Vitamin D receptor activators can • protect against vascular calcification. J Am Soc Nephrol 2008; 19:1509- 1519. The effect of vitamin D receptor activators on vascular calcification depends on their dosage. Low doses can be protective by inhibiting PTH secretion.
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J Am Soc Nephrol
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Mathew, S.1
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33845700085
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Levin A, Bakris GL, Molitch M, et al Prevalence of abnormal serum vitamin D, • PTH, calcium, and phosphorus in patients with chronic kidney disease: results of the study to evaluate early kidney disease. Kidney Int 2007; 71:31 -38. This study of CKD patients before dialysis gives reliable estimates of mineral metabolism parameters in a large unreferred population.
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Levin A, Bakris GL, Molitch M, et al Prevalence of abnormal serum vitamin D, • PTH, calcium, and phosphorus in patients with chronic kidney disease: results of the study to evaluate early kidney disease. Kidney Int 2007; 71:31 -38. This study of CKD patients before dialysis gives reliable estimates of mineral metabolism parameters in a large unreferred population.
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18
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41449084839
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Gutierrez OM, Isakova T, Andress DL, et el. Prevalence and severity of disordered mineral metabolism in Blacks with chronic kidney disease. Kidney Int 2008; 73:956-962.
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Gutierrez OM, Isakova T, Andress DL, et el. Prevalence and severity of disordered mineral metabolism in Blacks with chronic kidney disease. Kidney Int 2008; 73:956-962.
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19
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Andress DL, Coyne DW, Kalantar-Zadeh K, et al Management of secondary hyperparathyroidism in stages 3 and 4 chronic kidney disease. Endocr Pract 2008; 14:18-27.
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Andress, D.L.1
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Kalantar-Zadeh, K.3
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20
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Fibroblast growth factor-23 mitigates hyperphosphatemia but accentuates calcitriol deficiency in chronic kidney disease
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Gutierrez O, Isakova T, Rhee E, et al Fibroblast growth factor-23 mitigates hyperphosphatemia but accentuates calcitriol deficiency in chronic kidney disease. J Am Soc Nephrol 2005; 16:2205-2215.
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Gutierrez, O.1
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Relationship between vitamin D status, parathyroid hormone levels and bone mineral density in patients with chronic kidney disease stages 3 and 4
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Stavroulopoulos A, Porter CJ, Roe SD, et al Relationship between vitamin D status, parathyroid hormone levels and bone mineral density in patients with chronic kidney disease stages 3 and 4. Nephrology (Carlton) 2008; 13:63-67.
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Stavroulopoulos, A.1
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33745210682
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Survival predictability of timevarying indicators of bone disease in maintenance hemodialysis patients
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Kalantar-Zadeh K, Kuwae N, Regidor DL, et al Survival predictability of timevarying indicators of bone disease in maintenance hemodialysis patients. Kidney Int 2006; 70:771 -780.
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Kalantar-Zadeh, K.1
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39349107502
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Baber U, de Lemos JA, Khera A, et al Nontraditional risk factors predict coronary calcification in chronic kidney disease in a population-based cohort. Kidney Int 2008; 73:615-621.
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Baber, U.1
de Lemos, J.A.2
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38849167379
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Lanthanum carbonate effectively controls serum phosphate without affecting serum calcium levels in patients undergoing hemodialysis
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Shigematsu T. Lanthanum carbonate effectively controls serum phosphate without affecting serum calcium levels in patients undergoing hemodialysis. Ther Apher Dial 2008; 12:55-61.
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Shigematsu, T.1
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Effects of sevelamer and calciumbased phosphate binders on mortality in hemodialysis patients
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Suki WN, Zabaneh R, Cangiano JL, et al Effects of sevelamer and calciumbased phosphate binders on mortality in hemodialysis patients. Kidney Int 2007; 72:1130-1137.
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Suki, W.N.1
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Sevelamer attenuates the progression of coronary and aortic calcification in hemodialysis patients
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Chertow GM, Burke SK, Raggi P. Sevelamer attenuates the progression of coronary and aortic calcification in hemodialysis patients. Kidney Int 2002; 62:245-252.
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Chertow, G.M.1
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Raggi, P.3
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31
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38449117963
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Meta-analysis: Vitamin D •• compounds in chronic kidney disease
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This very carefully done metaanalysis shows that the largely used vitamin D compounds are prescribed on fragile grounds and call for more trials based on patient-level outcomes
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Palmer SC, McGregor DO, Macaskill P, et al Meta-analysis: vitamin D •• compounds in chronic kidney disease. Ann Intern Med 2007; 147:840- 853. This very carefully done metaanalysis shows that the largely used vitamin D compounds are prescribed on fragile grounds and call for more trials based on patient-level outcomes.
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Palmer, S.C.1
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An assessment of cinacalcet HCl effects on bone histology in dialysis patients with secondary hyperparathyroidism
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Effect of lowering dialysate calcium on bone and •• mineral parameters related to adynamic bone disease
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author reply 457. This study suggests that using dialysates with a low-calcium concentration could improve dialysis patients with a dynamic bone disease by stimulating PTH secretion
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Spasovski G, Vanholder R. Effect of lowering dialysate calcium on bone and •• mineral parameters related to adynamic bone disease. Ther Apher Dial 2007; 11:455-456; author reply 457. This study suggests that using dialysates with a low-calcium concentration could improve dialysis patients with a dynamic bone disease by stimulating PTH secretion.
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Spasovski, G.1
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Grobner T. Gadolinium-a specific trigger for the development of nephrogenic fibrosing dermopathy and nephrogenic systemic fibrosis? Nephrol Dial Transplant 2006; 21:1104-1108.
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Cutaneous changes of nephrogenic • systemic fibrosis: Predictor of early mortality and association with gadolinium exposure
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This interesting study of a cohort of haemodialysis patients implies gadopentate dimeglumine as a cause of NSF and shows that NSF is associated with increased short-term mortality
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Todd DJ, Kagan A, Chibnik LB, Kay J. Cutaneous changes of nephrogenic • systemic fibrosis: predictor of early mortality and association with gadolinium exposure. Arthritis Rheum 2007; 56:3433-3441. This interesting study of a cohort of haemodialysis patients implies gadopentate dimeglumine as a cause of NSF and shows that NSF is associated with increased short-term mortality.
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Arthritis Rheum
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Todd, D.J.1
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Nephrogenic systemic fibrosis: Risk factors and incidence estimation
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Sadowski EA, Bennett LK, Chan MR, et al. Nephrogenic systemic fibrosis: risk factors and incidence estimation. Radiology 2007; 243:148-157.
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Radiology
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Sadowski, E.A.1
Bennett, L.K.2
Chan, M.R.3
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54
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Gadolinium is not the only trigger for nephrogenic systemic fibrosis: Insights from two cases and review of the recent literature
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Wahba IM, Simpson EL, White K. Gadolinium is not the only trigger for nephrogenic systemic fibrosis: insights from two cases and review of the recent literature. Am J Transplant 2007; 7:2425-2432.
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Wahba, I.M.1
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Extracellular gadolinium contrast agents: Differences in stability
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Morcos SK. Extracellular gadolinium contrast agents: differences in stability. Eur J Radiol 2008; 66:175-179.
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Morcos, S.K.1
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Gadodiamide contrast agent •• 'activates' fibroblasts: A possible cause of nephrogenic systemic fibrosis
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This experimental study brings evidence that gadoliamide is a cause of NSF
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Edward M, Quinn JA, Mukherjee S, et al. Gadodiamide contrast agent •• 'activates' fibroblasts: a possible cause of nephrogenic systemic fibrosis. J Pathol 2008; 214:584-593. This experimental study brings evidence that gadoliamide is a cause of NSF.
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Edward, M.1
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Preclinical investigation to compare different gadolinium-based contrast agents regarding their propensity to release gadolinium in vivo and to trigger nephrogenic systemic fibrosis-like lesions
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Sieber MA, Lengsfeld P, Frenzel T, et al Preclinical investigation to compare different gadolinium-based contrast agents regarding their propensity to release gadolinium in vivo and to trigger nephrogenic systemic fibrosis-like lesions. Eur Radiol 2008; 18:2164-2173.
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Sieber, M.A.1
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Nephrogenic systemic fibrosis developed after recovery from acute renal failure: Gadolinium as a possible aetiological factor
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Epub ahead of print
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Lu CF, Hsiao CH, Tjiu JW. Nephrogenic systemic fibrosis developed after recovery from acute renal failure: gadolinium as a possible aetiological factor. J Eur Acad Dermatol Venereol 2008 [Epub ahead of print].
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Lu, C.F.1
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59
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High prevalence of nephrogenic • • systemic fibrosis in chronic renal failure patients exposed to gadodiamide, a gadolinium-containing magnetic resonance contrast agent
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An interesting study that documents the risk of NSF as a function of renal impairment and gadolinium exposure
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Rydahl C, Thomson HS, Marckmann P. High prevalence of nephrogenic • • systemic fibrosis in chronic renal failure patients exposed to gadodiamide, a gadolinium-containing magnetic resonance contrast agent. Invest Radiol 2008;43:141-144. An interesting study that documents the risk of NSF as a function of renal impairment and gadolinium exposure.
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Invest Radiol
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Rydahl, C.1
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Cardiac and vascular metal deposition with high mortality in nephrogenic systemic fibrosis
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Hair and fingernail gadolinium ICP-MS contents in an overdose case associated with nephrogenic systemic fibrosis
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Saussereau E, Lacroix C, Cattaneo A, et al. Hair and fingernail gadolinium ICP-MS contents in an overdose case associated with nephrogenic systemic fibrosis. Forensic Sci Int 2008; 176:54-57.
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Golding LP, Provenzale JM. Nephrogenic systemic fibrosis: possible association with a predisposing infection. AJR Am J Roentgenol 2008; 190:1069- 1075.
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AJR Am J Roentgenol
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Swaminathan S, Horn TD, Pellowski D, et al Nephrogenic systemic fibrosis, gadolinium, and iron mobilization. N Engl J Med 2007; 357:720-722.
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Kallen AJ, Jhung MA, Cheng S, et al Gadolinium-containing magnetic resonance imaging contrast and nephrogenic systemic fibrosis: a casecontrol study. Am J Kidney Dis 2008; 51:966-975.
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Marckmann P, Skov L, Rossen K, et al Case-control study of gadodiamiderelated nephrogenic systemic fibrosis. Nephrol Dial Transplant 2007; 22:3174-3178.
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An interesting case report with instructive discussion and literature review
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Kay J, Bazari H, Avery LL, Koreishi AF. Case 6-2008: a 46-year-old woman • with renal failure and stiffness of the joints and skin. N Engl J Med 2008; 358:827-838. An interesting case report with instructive discussion and literature review.
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Kay, J.1
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Successful pregnancy in a hemodialysis patient and marked resolution of her nephrogenic systemic fibrosis
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Khurana A, Nickel AE, Greene JF Jr, et al Successful pregnancy in a hemodialysis patient and marked resolution of her nephrogenic systemic fibrosis. Am J Kidney Dis 2008; 51 :e29-e32.
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Possibly enhanced Gd excretion in dialysate, but no major clinical benefit of 3-5 months of treatment with sodium thiosulfate in late stages of nephrogenic systemic fibrosis
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Marckmann P, Nielsen AH, Sloth JJ. Possibly enhanced Gd excretion in dialysate, but no major clinical benefit of 3-5 months of treatment with sodium thiosulfate in late stages of nephrogenic systemic fibrosis. Nephrol Dial Transplant 2008; 23:3280-3282.
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Perazella MA, Reilly RF. Nephrogenic systemic fibrosis: recommendations for gadolinium-based contrast use in patients with kidney disease. Semin Dial 2008; 21:171-173.
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Perazella, M.A.1
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