-
1
-
-
0032971161
-
Caspase-independent programmed cell death with necrotic morphology
-
Kitanaka C., and Kuchino Y. Caspase-independent programmed cell death with necrotic morphology. Cell Death Differ. 6 (1999) 508-515
-
(1999)
Cell Death Differ.
, vol.6
, pp. 508-515
-
-
Kitanaka, C.1
Kuchino, Y.2
-
2
-
-
0015880897
-
The morphology of various types of cell death in prenatal tissues
-
Schweichel J.U., and Merker H.J. The morphology of various types of cell death in prenatal tissues. Teratology 7 (1973) 253-266
-
(1973)
Teratology
, vol.7
, pp. 253-266
-
-
Schweichel, J.U.1
Merker, H.J.2
-
3
-
-
33749178260
-
Necrosis, a well-orchestrated form of cell demise: signalling cascades, important mediators and concomitant immune response
-
Festjens N., et al. Necrosis, a well-orchestrated form of cell demise: signalling cascades, important mediators and concomitant immune response. Biochim. Biophys. Acta 1757 (2006) 1371-1387
-
(2006)
Biochim. Biophys. Acta
, vol.1757
, pp. 1371-1387
-
-
Festjens, N.1
-
4
-
-
33846018602
-
Cell death by necrosis: towards a molecular definition
-
Golstein P., and Kroemer G. Cell death by necrosis: towards a molecular definition. Trends Biochem. Sci. 32 (2007) 37-43
-
(2007)
Trends Biochem. Sci.
, vol.32
, pp. 37-43
-
-
Golstein, P.1
Kroemer, G.2
-
5
-
-
29944438881
-
Necrotic death as a cell fate
-
Zong W.X., and Thompson C.B. Necrotic death as a cell fate. Genes Dev. 20 (2006) 1-15
-
(2006)
Genes Dev.
, vol.20
, pp. 1-15
-
-
Zong, W.X.1
Thompson, C.B.2
-
6
-
-
0032494143
-
Dual signaling of the Fas receptor: initiation of both apoptotic and necrotic cell death pathways
-
Vercammen D., et al. Dual signaling of the Fas receptor: initiation of both apoptotic and necrotic cell death pathways. J. Exp. Med. 188 (1998) 919-930
-
(1998)
J. Exp. Med.
, vol.188
, pp. 919-930
-
-
Vercammen, D.1
-
7
-
-
33644840693
-
Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury
-
Degterev A., et al. Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury. Nat. Chem. Biol. 1 (2005) 112-119
-
(2005)
Nat. Chem. Biol.
, vol.1
, pp. 112-119
-
-
Degterev, A.1
-
8
-
-
5944233768
-
Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule
-
Holler N., et al. Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule. Nat. Immunol. 1 (2000) 489-495
-
(2000)
Nat. Immunol.
, vol.1
, pp. 489-495
-
-
Holler, N.1
-
9
-
-
42249102086
-
Identification of RIP1 kinase as a specific cellular target of necrostatins
-
Degterev A., et al. Identification of RIP1 kinase as a specific cellular target of necrostatins. Nat. Chem. Biol. 4 (2008) 313-321
-
(2008)
Nat. Chem. Biol.
, vol.4
, pp. 313-321
-
-
Degterev, A.1
-
10
-
-
36849056027
-
The cardioprotective effect of necrostatin requires the cyclophilin-D component of the mitochondrial permeability transition pore
-
Lim S.Y., et al. The cardioprotective effect of necrostatin requires the cyclophilin-D component of the mitochondrial permeability transition pore. Cardiovasc. Drugs Ther. 21 (2007) 467-469
-
(2007)
Cardiovasc. Drugs Ther.
, vol.21
, pp. 467-469
-
-
Lim, S.Y.1
-
11
-
-
34548564715
-
Necrostatin: a potentially novel cardioprotective agent?
-
Smith C.C., et al. Necrostatin: a potentially novel cardioprotective agent?. Cardiovasc. Drugs Ther. 21 (2007) 227-233
-
(2007)
Cardiovasc. Drugs Ther.
, vol.21
, pp. 227-233
-
-
Smith, C.C.1
-
12
-
-
14744299357
-
The RIP kinases: crucial integrators of cellular stress
-
Meylan E., and Tschopp J. The RIP kinases: crucial integrators of cellular stress. Trends Biochem. Sci. 30 (2005) 151-159
-
(2005)
Trends Biochem. Sci.
, vol.30
, pp. 151-159
-
-
Meylan, E.1
Tschopp, J.2
-
13
-
-
0242663910
-
The death domain kinase RIP1 is essential for tumor necrosis factor α signaling to p38 mitogen-activated protein kinase
-
Lee T.H., et al. The death domain kinase RIP1 is essential for tumor necrosis factor α signaling to p38 mitogen-activated protein kinase. Mol. Cell. Biol. 23 (2003) 8377-8385
-
(2003)
Mol. Cell. Biol.
, vol.23
, pp. 8377-8385
-
-
Lee, T.H.1
-
14
-
-
0008206988
-
TRAF2 is essential for JNK but not NF-κB activation and regulates lymphocyte proliferation and survival
-
Lee S.Y., et al. TRAF2 is essential for JNK but not NF-κB activation and regulates lymphocyte proliferation and survival. Immunity 7 (1997) 703-713
-
(1997)
Immunity
, vol.7
, pp. 703-713
-
-
Lee, S.Y.1
-
15
-
-
0042090891
-
The role of the death-domain kinase RIP in tumour-necrosis-factor-induced activation of mitogen-activated protein kinases
-
Devin A., et al. The role of the death-domain kinase RIP in tumour-necrosis-factor-induced activation of mitogen-activated protein kinases. EMBO Rep. 4 (2003) 623-627
-
(2003)
EMBO Rep.
, vol.4
, pp. 623-627
-
-
Devin, A.1
-
16
-
-
4043136609
-
The kinase activity of Rip1 is not required for tumor necrosis factor-α-induced IκB kinase or p38 MAP kinase activation or for the ubiquitination of Rip1 by Traf2
-
Lee T.H., et al. The kinase activity of Rip1 is not required for tumor necrosis factor-α-induced IκB kinase or p38 MAP kinase activation or for the ubiquitination of Rip1 by Traf2. J. Biol. Chem. 279 (2004) 33185-33191
-
(2004)
J. Biol. Chem.
, vol.279
, pp. 33185-33191
-
-
Lee, T.H.1
-
17
-
-
33847051539
-
RIP1, a kinase on the crossroads of a cell's decision to live or die
-
Festjens N., et al. RIP1, a kinase on the crossroads of a cell's decision to live or die. Cell Death Differ. 14 (2007) 400-410
-
(2007)
Cell Death Differ.
, vol.14
, pp. 400-410
-
-
Festjens, N.1
-
18
-
-
0032033132
-
The death domain kinase RIP mediates the TNF-induced NF-κB signal
-
Kelliher M.A., et al. The death domain kinase RIP mediates the TNF-induced NF-κB signal. Immunity 8 (1998) 297-303
-
(1998)
Immunity
, vol.8
, pp. 297-303
-
-
Kelliher, M.A.1
-
19
-
-
0033214236
-
Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis
-
Lin Y., et al. Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis. Genes Dev. 13 (1999) 2514-2526
-
(1999)
Genes Dev.
, vol.13
, pp. 2514-2526
-
-
Lin, Y.1
-
20
-
-
34249820757
-
TNF-induced activation of the Nox1 NADPH oxidase and its role in the induction of necrotic cell death
-
Kim Y.S., et al. TNF-induced activation of the Nox1 NADPH oxidase and its role in the induction of necrotic cell death. Mol. Cell 26 (2007) 675-687
-
(2007)
Mol. Cell
, vol.26
, pp. 675-687
-
-
Kim, Y.S.1
-
21
-
-
34247264421
-
Structure-activity relationship study of tricyclic necroptosis inhibitors
-
Jagtap P.G., et al. Structure-activity relationship study of tricyclic necroptosis inhibitors. J. Med. Chem. 50 (2007) 1886-1895
-
(2007)
J. Med. Chem.
, vol.50
, pp. 1886-1895
-
-
Jagtap, P.G.1
-
22
-
-
33846913723
-
Structure-activity relationship analysis of a novel necroptosis inhibitor, Necrostatin-5
-
Wang K., et al. Structure-activity relationship analysis of a novel necroptosis inhibitor, Necrostatin-5. Bioorg. Med. Chem. Lett. 17 (2007) 1455-1465
-
(2007)
Bioorg. Med. Chem. Lett.
, vol.17
, pp. 1455-1465
-
-
Wang, K.1
-
23
-
-
34547154729
-
Activation segment exchange: a common mechanism of kinase autophosphorylation?
-
Oliver A.W., et al. Activation segment exchange: a common mechanism of kinase autophosphorylation?. Trends Biochem. Sci. 32 (2007) 351-356
-
(2007)
Trends Biochem. Sci.
, vol.32
, pp. 351-356
-
-
Oliver, A.W.1
-
24
-
-
0034675919
-
Activation of B-Raf kinase requires phosphorylation of the conserved residues Thr598 and Ser601
-
Zhang B.H., and Guan K.L. Activation of B-Raf kinase requires phosphorylation of the conserved residues Thr598 and Ser601. EMBO J. 19 (2000) 5429-5439
-
(2000)
EMBO J.
, vol.19
, pp. 5429-5439
-
-
Zhang, B.H.1
Guan, K.L.2
-
25
-
-
4444341939
-
Compartmentalization of TNF receptor 1 signaling: internalized TNF receptosomes as death signaling vesicles
-
Schneider-Brachert W., et al. Compartmentalization of TNF receptor 1 signaling: internalized TNF receptosomes as death signaling vesicles. Immunity 21 (2004) 415-428
-
(2004)
Immunity
, vol.21
, pp. 415-428
-
-
Schneider-Brachert, W.1
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