Activation of protein kinase Cα signaling prevents cytotoxicity and mutagenicity following lead acetate in CL3 human lung cancer cells

Toxicology

Volumn 250, Issue 1, 2008, Pages 55-61

  Wang, Chun Yu ^a   Lin, Yun Wei ^a   Yang, Jia Ling ^a  

^a BEIJING INSTITUTE OF BIOTECHNOLOGY   (China)

Author keywords

Lead acetate; Lung cancer cells; Mutation frequency; Phospho Raf 1S338; PKC

Indexed keywords

LEAD ACETATE; PROTEIN KINASE C ALPHA;

ARTICLE; CANCER CELL; CONTROLLED STUDY; CYTOTOXICITY; ENZYME ACTIVITY; HUMAN; HUMAN CELL; LUNG CANCER; MUTAGENICITY; PRIORITY JOURNAL;

ADENOCARCINOMA; CELL LINE, TUMOR; HUMANS; HYPOXANTHINE PHOSPHORIBOSYLTRANSFERASE; LUNG NEOPLASMS; MITOGEN-ACTIVATED PROTEIN KINASE 1; MITOGEN-ACTIVATED PROTEIN KINASE 3; MUTAGENESIS; MUTAGENS; ORGANOMETALLIC COMPOUNDS; PHOSPHORYLATION; PROTEIN KINASE C-ALPHA; PROTEIN TRANSPORT; PROTO-ONCOGENE PROTEINS C-RAF; SIGNAL TRANSDUCTION;

EID: 47849098786     PISSN: 0300483X     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.tox.2008.06.001     Document Type: Article

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