Carbon monoxide-releasing molecule tricarbonyldichlororuthenium (II) dimer induces concentration-dependent alterations in the electrophysiological properties of axons in mammalian spinal cord

Neuroscience

Volumn 151, Issue 4, 2008, Pages 1104-1111

  Davies, A L ^a,b   Kramer, J L K ^a,b   Hayes, K C ^a,b  

^a UNIVERSITY OF WESTERN ONTARIO   (Canada)

^b LAWSON HEALTH RESEARCH INSTITUTE   (Canada)

Author keywords

double sucrose gap; guinea pig; spinal cord injury

Indexed keywords

CARBON MONOXIDE; RUTHENIUM DERIVATIVE; TRICARBONYLDICHLORORUTHENIUM; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ELECTROPHYSIOLOGY; FEMALE; GUINEA PIG; MEMBRANE POTENTIAL; NERVE CONDUCTION; NEUROTOXICITY; NONHUMAN; PRIORITY JOURNAL; SPINAL CORD;

ANIMALS; AXONS; DOSE-RESPONSE RELATIONSHIP, DRUG; DOSE-RESPONSE RELATIONSHIP, RADIATION; ELECTRIC STIMULATION; GUINEA PIGS; MEMBRANE POTENTIALS; ORGANOMETALLIC COMPOUNDS; SPINAL CORD;

EID: 39149112486     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2007.12.002     Document Type: Article

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