Hypoxia suppresses astrocyte glutamate transport independently of amyloid formation

Biochemical and Biophysical Research Communications

Volumn 364, Issue 1, 2007, Pages 100-104

  Boycott, Hannah E ^a   Dallas, Mark ^a   Boyle, John P ^a   Pearson, Hugh A ^a   Peers, Chris ^a  

^a UNIVERSITY OF LEEDS   (United Kingdom)

Author keywords

Alzheimer's; Amyloid; Glia; Glutamate transport; Hypoxia

Indexed keywords

AMYLOID; AMYLOID BETA PROTEIN; BETA SECRETASE; EAAT1 PROTEIN; EAAT2 PROTEIN; GAMMA SECRETASE; GLUTAMATE TRANSPORTER; GLUTAMIC ACID; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; AMINO ACID TRANSPORT; ANIMAL CELL; ANIMAL TISSUE; ARTICLE; ASTROCYTE; CELL DEATH; CONTROLLED STUDY; HOMEOSTASIS; HYPOXIA; NERVE CELL LESION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT;

AMYLOID; AMYLOID BETA-PROTEIN; ANIMALS; ANOXIA; ASTROCYTES; BIOLOGICAL TRANSPORT; CELLS, CULTURED; EVOKED POTENTIALS; GLUTAMIC ACID; RATS; RATS, WISTAR;

EID: 35349002281     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2007.09.102     Document Type: Article

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