Hypoxia potentiates exocytosis and Ca2+ channels in PC12 cells via increased amyloid β peptide formation and reactive oxygen species generation

Journal of Physiology

Volumn 541, Issue 3, 2002, Pages 1013-1023

  Green, Kim N ^a   Boyle, John P ^a   Peers, Chris ^a  

^a UNIVERSITY OF LEEDS   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; ANTIOXIDANT; ASCORBIC ACID; CADMIUM; CALCIUM CHANNEL L TYPE; CALCIUM ION; CATECHOLAMINE; EBSELEN; GAMMA SECRETASE; HYDROGEN PEROXIDE; MELATONIN; MONOCLONAL ANTIBODY; REACTIVE OXYGEN METABOLITE; CALCIUM CHANNEL; GAMMA SECRETASE INHIBITOR;

ALZHEIMER DISEASE; AMINO TERMINAL SEQUENCE; AMPEROMETRY; ANIMAL CELL; ARTICLE; CALCIUM CURRENT; CALCIUM HOMEOSTASIS; CALCIUM TRANSPORT; CATECHOLAMINE RELEASE; CELL STRAIN; CONTROLLED STUDY; EXOCYTOSIS; EXPOSURE; HYPOXIA; IMMUNOFLUORESCENCE; NONHUMAN; PATCH CLAMP; PHEOCHROMOCYTOMA CELL; PRIORITY JOURNAL; RECEPTOR UPREGULATION; HYPOXIC CELL; PC12 CELL LINE; PEPTIDE SYNTHESIS; PROTEIN FUNCTION; RAT; UPREGULATION; WHOLE CELL PATCH CLAMP;

AMYLOID BETA-PROTEIN; ANIMALS; ANOXIA; ANTIOXIDANTS; ASCORBIC ACID; CADMIUM; CALCIUM CHANNELS; ELECTROPHYSIOLOGY; EXOCYTOSIS; IMMUNOHISTOCHEMISTRY; MELATONIN; MEMBRANE POTENTIALS; MICROELECTRODES; OXIDATIVE STRESS; PATCH-CLAMP TECHNIQUES; PC12 CELLS; RATS; REACTIVE OXYGEN SPECIES;

EID: 0037098737     PISSN: 00223751     EISSN: None     Source Type: Journal    
DOI: 10.1113/jphysiol.2002.017582     Document Type: Article

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