Granulocyte colony-stimulating factor activates Wnt signal to sustain gap junction function through recruitment of β-catenin and cadherin

FEBS Letters

Volumn 581, Issue 25, 2007, Pages 4821-4830

  Kuwabara, Masanori ^a   Kakinuma, Yoshihiko ^a   Katare, Rajesh G ^a   Ando, Motonori ^b   Yamasaki, Fumiyasu ^a   Doi, Yoshinori ^a   Sato, Takayuki ^a  

^a KOCHI MEDICAL SCHOOL   (Japan)

^b OKAYAMA UNIVERSITY   (Japan)

Author keywords

Catenin; Cadherin; Connexin; Gap junction; Granulocyte colony stimulating factor; Hypoxia

Indexed keywords

BETA CATENIN; CADHERIN; GRANULOCYTE COLONY STIMULATING FACTOR;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL COMMUNICATION; CELL MEMBRANE; CONTROLLED STUDY; DRUG MECHANISM; GAP JUNCTION; HEART ARRHYTHMIA; HEART INFARCTION; HEART MUSCLE CELL; HEART VENTRICLE ARRHYTHMIA; HYPOXIA; NONHUMAN; PHOSPHORYLATION; PRIORITY JOURNAL; RAT; UPREGULATION;

ANIMALS; ANTI-ARRHYTHMIA AGENTS; BETA CATENIN; CADHERINS; CELL HYPOXIA; CELL LINE; CELL SURVIVAL; CELLS, CULTURED; CONNEXIN 43; GAP JUNCTIONS; GRANULOCYTE COLONY-STIMULATING FACTOR; MALE; MYOCARDIAL INFARCTION; MYOCYTES, CARDIAC; RATS; RATS, WISTAR; RECEPTORS, GRANULOCYTE COLONY-STIMULATING FACTOR; SIGNAL TRANSDUCTION; WNT PROTEINS;

EID: 34948819272     PISSN: 00145793     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.febslet.2007.09.007     Document Type: Article

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