Slow Conduction and Enhanced Anisotropy Increase the Propensity for Ventricular Tachyarrhythmias in Adult Mice with Induced Deletion of Connexin43

Circulation

Volumn 109, Issue 8, 2004, Pages 1048-1055

  Van Rijen, Harold V M ^a   Eckardt, Dominik ^b   Degen, Joachim ^b   Theis, Martin ^b,d   Ott, Thomas ^b   Willecke, Klaus ^b   Jongsma, Habo J ^a   Opthof, Tobias ^b   De Bakker, Jacques M T ^a,c  

^a UNIVERSITY MEDICAL CENTER UTRECHT   (Netherlands)

^b UNIVERSITY OF BONN   (Germany)

^c ROYAL NETHERLANDS ACADEMY OF ARTS AND SCIENCES   (Netherlands)

^d Och Spine at New York Presbyterian Hospitals   (United States)

Author keywords

Conduction; Mapping; Tachyarrhythmias

Indexed keywords

4 HYDROXYTAMOXIFEN; CONNEXIN 43; HYBRID PROTEIN;

ALLELE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANISOTROPY; ARTICLE; CONTROLLED STUDY; EPICARDIUM; GENE DELETION; GENE FUSION; GENETIC CODE; HEART CONDUCTION; HEART MUSCLE CONDUCTION SYSTEM; HEART PERFUSION; HEART VENTRICLE CONDUCTION; HEART VENTRICLE TACHYCARDIA; ISOLATED HEART; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SINUS RHYTHM;

ANIMALS; ANISOTROPY; COLLAGEN; CONNEXIN 43; GENOTYPE; HEART CONDUCTION SYSTEM; MICE; MICE, KNOCKOUT; NEURAL CONDUCTION; TACHYCARDIA, VENTRICULAR; VENTRICULAR PREMATURE COMPLEXES;

EID: 1442359119     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.0000117402.70689.75     Document Type: Article

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