Heat shock does not induce γH2AX foci formation but protects cells from N-methyl-N′-nitro-N-nitrosoguanidine-induced genotoxicity

Mutation Research - Genetic Toxicology and Environmental Mutagenesis

Volumn 629, Issue 1, 2007, Pages 40-48

  Dong, Zhengwei ^a   Hu, Hu ^b   Chen, Weijun ^b   Li, Zhongxiang ^b   Liu, Guangyi ^a   Yang, Jun ^a,b  

^a ZHEJIANG UNIVERSITY   (China)

^b ZHEJIANG UNIVERSITY SCHOOL OF MEDICINE   (China)

Author keywords

H2AX; DNA damage; Double strand breaks; Heat shock

Indexed keywords

FLAP ENDONUCLEASE; HISTONE H2AX; METHYLNITRONITROSOGUANIDINE; PROTEIN P53;

ANIMAL CELL; ARTICLE; CELL STRAIN HEPG2; CELL VIABILITY; CONTROLLED STUDY; DNA STRAND BREAKAGE; EMBRYO; GENE INDUCTION; GENOTOXICITY; HEAT SHOCK; HELA CELL; HUMAN; HUMAN CELL; MALE; NONHUMAN; PRIORITY JOURNAL; SPERMATOZOON;

CARCINOGENS; CARCINOMA, HEPATOCELLULAR; CELL LINE; CELL LINE, TUMOR; CELL SURVIVAL; DNA; DNA DAMAGE; DNA, NEOPLASM; FLOW CYTOMETRY; FLUORESCEIN-5-ISOTHIOCYANATE; FLUORESCENT ANTIBODY TECHNIQUE, DIRECT; FLUORESCENT DYES; HEAT; HELA CELLS; HISTONES; HUMANS; INDOLES; LIVER NEOPLASMS; MALE; METHYLNITRONITROSOGUANIDINE; MICROSCOPY, FLUORESCENCE; SPERMATOZOA;

EID: 33947582985     PISSN: 13835718     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mrgentox.2007.01.003     Document Type: Article

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