Disease-associated casein kinase I δ mutation may promote adenomatous polyps formation via a Wnt/β-catenin independent mechanism

International Journal of Cancer

Volumn 120, Issue 5, 2007, Pages 1005-1012

  Tsai, I Chun ^a   Woolf, Margaret ^a   Neklason, Deborah W ^a   Branford, William W ^a,b   Yost, H Joseph ^a   Burt, Randall W ^a   Virshup, David M ^a,c  

^a UNIVERSITY OF UTAH SCHOOL OF MEDICINE   (United States)

^b WAYNE STATE UNIVERSITY   (United States)

^c UNIVERSITY OF UTAH   (United States)

Author keywords

catenin; Adenomatous polyps; Casein kinase I; Noncanonical Wnt pathway; Wnt pathway

Indexed keywords

ARGININE; BETA CATENIN; CASEIN KINASE IDELTA; CASEIN KINASE IEPSILON; HISTIDINE; WNT PROTEIN;

ADENOMATOUS POLYP; ANIMAL CELL; ARTICLE; CANCER GROWTH; CANCER RISK; CELL TRANSFORMATION; COLON ADENOMA; COLON CANCER; COLORECTAL CANCER; CONTROLLED STUDY; DNA SEQUENCE; EMBRYO DEVELOPMENT; ENZYME ACTIVITY; ENZYME ASSAY; EXON; FAMILIAL CANCER; GENE MUTATION; HUMAN; HUMAN CELL; IN VITRO STUDY; MISSENSE MUTATION; MORPHOLOGY; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; UPREGULATION; WILD TYPE; XENOPUS;

ADENOMATOUS POLYPS; AMINO ACID SEQUENCE; AMINO ACID SUBSTITUTION; ANIMALS; ARGININE; BETA CATENIN; CASEIN KINASE IDELTA; COLONIC NEOPLASMS; EXONS; HETEROZYGOTE; HISTIDINE; HUMANS; MOLECULAR SEQUENCE DATA; MUTATION; PEDIGREE; WNT PROTEINS; XENOPUS;

EID: 33846827050     PISSN: 00207136     EISSN: 10970215     Source Type: Journal    
DOI: 10.1002/ijc.22368     Document Type: Article

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