Functional impairment of cardiac transient outward K+ current as a result of abnormally altered cellular environment

Clinical and Experimental Pharmacology and Physiology

Volumn 34, Issue 3, 2007, Pages 148-152

  Du, Zhimin ^a,b   Chaoqian, Xu ^c   Shan, Hongli ^c   Lu, Yanjie ^c   Ren, Nanqi ^a  

^a HARBIN INSTITUTE OF TECHNOLOGY   (China)

^b HARBIN MEDICAL UNIVERSITY   (China)

^c Department of Pharmacology and Bio pharmaceutical   (China)

Author keywords

Acidosis; Cellular environment; Ito; Ischaemia; Nitrogen gas

Indexed keywords

ACIDOSIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CELL FUNCTION; CONTROLLED STUDY; CORONARY ARTERY LIGATION; FEMALE; HEART ARRHYTHMIA; HEART DISEASE; HEART ELECTROPHYSIOLOGY; HEART MUSCLE CELL; HEART MUSCLE CONDUCTION DISTURBANCE; HEART MUSCLE ISCHEMIA; HEART MUSCLE POTENTIAL; HEART REPOLARIZATION; MALE; NONHUMAN; POTASSIUM CURRENT; STRESS;

ACIDOSIS; ALGORITHMS; ANIMALS; ANOXIA; CELL SEPARATION; DOGS; FEMALE; HYDROGEN-ION CONCENTRATION; ISCHEMIA; MALE; MYOCARDIAL ISCHEMIA; MYOCARDIUM; MYOCYTES, CARDIAC; NITROGEN; PATCH-CLAMP TECHNIQUES; POTASSIUM CHANNELS; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;

EID: 33846449849     PISSN: 03051870     EISSN: 14401681     Source Type: Journal    
DOI: 10.1111/j.1440-1681.2007.04527.x     Document Type: Article

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