Hyperoxia in the intensive care unit: Why more is not always better

Current Opinion in Critical Care

Volumn 13, Issue 1, 2007, Pages 73-78

  Altemeier, William A ^a   Sinclair, Scott E ^b  

^a UNIVERSITY OF WASHINGTON   (United States)

^b UNIVERSITY OF TENNESSEE HEALTH SCIENCE CENTER   (United States)

Author keywords

Apoptosis; ARDS; Mechanical ventilation; Reactive oxygen species; Respiratory failure

Indexed keywords

APOPTOTIC PROTEASE ACTIVATING FACTOR 1; CASPASE 9; COPPER ZINC SUPEROXIDE DISMUTASE; CYTOCHROME C; CYTOKINE; FAS ANTIGEN; GLUTATHIONE PEROXIDASE; HYDROGEN PEROXIDE; HYDROXYL RADICAL; MANGANESE SUPEROXIDE DISMUTASE; REACTIVE OXYGEN METABOLITE; SUPEROXIDE;

ACUTE LUNG INJURY; ADULT RESPIRATORY DISTRESS SYNDROME; APOPTOSIS; ASSISTED VENTILATION; CELL DEATH; CRITICALLY ILL PATIENT; CYTOKINE PRODUCTION; ENZYME ACTIVATION; HUMAN; HYPEROXIA; INNATE IMMUNITY; INTENSIVE CARE; LUNG MECHANICS; NONHUMAN; OXYGEN THERAPY; PATHOGENESIS; PROTEIN EXPRESSION; RESPIRATORY FAILURE; REVIEW; SIGNAL TRANSDUCTION; TIDAL VOLUME;

ANIMALS; APOPTOSIS; HUMANS; HYPEROXIA; INTENSIVE CARE UNITS; OXYGEN; REACTIVE OXYGEN SPECIES; RESPIRATION, ARTIFICIAL; RESPIRATORY DISTRESS SYNDROME, ADULT; RESPIRATORY INSUFFICIENCY; RISK FACTORS;

EID: 33846109765     PISSN: 10705295     EISSN: 15317072     Source Type: Journal    
DOI: 10.1097/MCC.0b013e32801162cb     Document Type: Review

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