Tissue kallikrein protects against pressure overload-induced cardiac hypertrophy through kinin B2 receptor and glycogen synthase kinase-3β activation

Cardiovascular Research

Volumn 73, Issue 1, 2007, Pages 130-142

  Li, Huey Jiun ^a   Yin, Hang ^a   Yao, Yu Yu ^a   Shen, Bo ^a   Bader, Michael ^b   Chao, Lee ^a   Chao, Julie ^a  

^a MEDICAL UNIVERSITY OF SOUTH CAROLINA   (United States)

^b MAX DELBRÜCK CENTER FOR MOLECULAR MEDICINE   (Germany)

Author keywords

Glycogen synthase kinase 3 ; Hypertrophy; Kinin B2 receptor; Nuclear factor B; Tissue kallikrein

Indexed keywords

GLYCOGEN SYNTHASE KINASE 3BETA; ICATIBANT; KALLIKREIN; KININ;

ADENOVIRUS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTA CONSTRICTION; ARTICLE; CONTROLLED STUDY; GENE TARGETING; HEART FUNCTION; HEART MUSCLE; HEART WEIGHT; MALE; NONHUMAN; PRIORITY JOURNAL; RAT;

ADENOVIRIDAE; ANIMALS; BLOTTING, WESTERN; CARDIOMEGALY; ELECTROPHORETIC MOBILITY SHIFT ASSAY; ENZYME ACTIVATION; GENE EXPRESSION; GENETIC VECTORS; GLYCOGEN SYNTHASE KINASE 3; HUMANS; ISOENZYMES; MALE; MICE; MICE, KNOCKOUT; NAD; NADP; NF-KAPPA B; NITRATES; RATS; RATS, WISTAR; RECEPTOR, BRADYKININ B1; RECEPTOR, BRADYKININ B2; SUPEROXIDES; TISSUE KALLIKREINS; TRANSDUCTION, GENETIC; TRANSGENES;

EID: 33845580042     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.cardiores.2006.10.014     Document Type: Article

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