Redox-sensitive intermediates mediate angiotensin II-induced p38 MAP kinase activation, AP-1 binding activity, and TGF-beta expression in adult ventricular cardiomyocytes.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 15, Issue 12, 2001, Pages 2291-2293

  Wenzel, S ^a   Taimor, G ^a   Piper, H M ^a   Schluter K D ^a  

^a JUSTUS LIEBIG UNIVERSITY   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

ANGIOTENSIN II; MESSENGER RNA; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE P38; TRANSCRIPTION FACTOR AP 1; TRANSFORMING GROWTH FACTOR BETA;

ANIMAL; ARTICLE; BIOLOGICAL MODEL; BIOSYNTHESIS; CARDIOMEGALY; CELL CULTURE; ENZYME ACTIVATION; GENETICS; HEART MUSCLE; HEART VENTRICLE; METABOLISM; OXIDATION REDUCTION REACTION; SIGNAL TRANSDUCTION; TRANSACTIVATION;

ANGIOTENSIN II; ANIMALS; CARDIOMEGALY; CELLS, CULTURED; ENZYME ACTIVATION; HEART VENTRICLES; MAP KINASE SIGNALING SYSTEM; MITOGEN-ACTIVATED PROTEIN KINASES; MODELS, BIOLOGICAL; MYOCARDIUM; OXIDATION-REDUCTION; P38 MITOGEN-ACTIVATED PROTEIN KINASES; RNA, MESSENGER; TRANS-ACTIVATION (GENETICS); TRANSCRIPTION FACTOR AP-1; TRANSFORMING GROWTH FACTOR BETA;

EID: 0035487975     PISSN: None     EISSN: 15306860     Source Type: Journal    
DOI: None     Document Type: Article

References (0)