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Identification of cell-of-origin breast tumor subtypes in inflammatory breast cancer by gene expression profiling
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Van Laere, S.J.1
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Gene expression profiles of multiple breast cancer phenotypes and response to neoadjuvant chemotherapy
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A novel putative low affinity insulin-like growth factor-binding protein LIBC (lost in inflammatory breast cancer), and RhoC GTPase correlate with the inflammatory breast cancer phenotype
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RhoC GTPase, a novel transforming oncogene for human mammary epithelial cells that partially recapitulates the inflammatory breast cancer phenotype
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WISP3 is a novel tumor suppressor gene of inflammatory breast cancer
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WISP3 and RhoC guanosine triphosphatase cooperate in the development of inflammatory breast cancer
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Overexpression of caveolin-1 and -2 in cell lines and in human samples of inflammatory breast cancer
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Van den Eynden GG, Van Laere SJ, Van der Auwera I, et al. Overexpression of caveolin-1 and -2 in cell lines and in human samples of inflammatory breast cancer. Breast Cancer Res Treat 2006; 95:219-228. The overexpression of either caveolin-1 or 2 is confirmed in cell lines and in human tissue samples of IBC. It is suggested to be caused by promotor hypomethylation.
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Nuclear factor-kappaB signature of inflammatory breast cancer by cDNA microarray validated by quantitative real-time reverse transcription-PCR, immunohistochemistry, and nuclear factor-kappaB DNA binding
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Identification of pathway-selective estrogen receptor ligands that inhibit NF-kappaB transcriptional activity
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A cytoplasmic substrate of mitogen-activated protein kinase is responsible for estrogen receptor-alpha downregulation in breast cancer cells: The role of nuclear factor-kappaB
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