Absence of endothelial cells, central necrosis, and fibrosis are associated with aggressive inflammatory breast cancer

Cancer Research

Volumn 61, Issue 2, 2001, Pages 445-451

  Shirakawa, Kazuo ^a,e   Tsuda, Hitoshi ^a   Heike, Yuji ^b   Kato, Kazunori ^a   Asada, Rumiko ^a   Inomata, Motoko ^a   Sasaki, Hiroki ^a   Kasumi, Fujio ^c   Yoshimoto, Masataka ^c   Iwanaga, Toshihiko ^d   Konishi, Fumio ^e   Terada, Masaaki ^a   Wakasugi, Hiro ^a  

^a NATIONAL CANCER CENTER RESEARCH INSTITUTE   (Japan)

^b NATIONAL HOSPITAL ORGANIZATION SHIKOKU CANCER CENTER   (Japan)

^c CANCER CHEMOTHERAPY CENTER   (Japan)

^d HOKKAIDO UNIVERSITY   (Japan)

^e JICHI MEDICAL UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

ANGIOPOIETIN; BASIC FIBROBLAST GROWTH FACTOR; CD31 ANTIGEN; EPIDERMAL GROWTH FACTOR; ESTROGEN RECEPTOR; INTEGRIN; INTERLEUKIN 8; PROGESTERONE RECEPTOR;

ANEUPLOIDY; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BREAST CANCER; CANCER CELL CULTURE; CANCER TRANSPLANTATION; CONTROLLED STUDY; DISEASE ASSOCIATION; ENDOTHELIUM CELL; ENZYME LINKED IMMUNOSORBENT ASSAY; ERYTHEMA; FEMALE; FIBROSIS; GENE AMPLIFICATION; GENE EXPRESSION; HISTOPATHOLOGY; HUMAN; HUMAN CELL; IMMUNOHISTOCHEMISTRY; MOUSE; NONHUMAN; PRIORITY JOURNAL; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SCID MOUSE; TISSUE NECROSIS; XENOGRAFT;

ANGIOGENESIS INDUCING AGENTS; ANIMALS; BREAST NEOPLASMS; CADHERINS; CELL DIVISION; CYTOKINES; ENDOTHELIUM, VASCULAR; ENZYME-LINKED IMMUNOSORBENT ASSAY; FEMALE; FIBROSIS; FLOW CYTOMETRY; GENE EXPRESSION REGULATION, NEOPLASTIC; HUMANS; IMMUNOHISTOCHEMISTRY; INTEGRINS; MICE; MICE, INBRED BALB C; MICE, NUDE; MICROSCOPY, ELECTRON; MIDDLE AGED; NECROSIS; NEOPLASM TRANSPLANTATION; RECEPTOR PROTEIN-TYROSINE KINASES; RECEPTOR, TIE-2; RECEPTORS, GROWTH FACTOR; RECEPTORS, VASCULAR ENDOTHELIAL GROWTH FACTOR; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; RNA, MESSENGER; TRANSPLANTATION, HETEROLOGOUS; TUMOR CELLS, CULTURED;

EID: 0035863521     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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