Activation of apoptotic pathways at muscle fiber synapses is circumscribed and reversible in a slow-channel syndrome model

Neurobiology of Disease

Volumn 23, Issue 2, 2006, Pages 462-470

  Vohra, Bhupinder P S ^b   Groshong, Jason S ^a   Zayas, Roberto ^a   Wollmann, Robert L ^c   Gomez, Christopher M ^d  

^a UNIVERSITY OF MINNESOTA   (United States)

^b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c UNIVERSITY OF CHICAGO MEDICAL CENTER   (United States)

^d AMB S237   (United States)

Author keywords

Acetylcholine receptor; Apoptosis; Caspase; Channelopathy; Mutation; Neuromuscular junction

Indexed keywords

CASPASE 3; CASPASE 9; CHOLINERGIC RECEPTOR; CYSTEINE PROTEINASE INHIBITOR; SINGLE STRANDED DNA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRAIN NUCLEUS; CHOLINERGIC ACTIVITY; CHROMATIN CONDENSATION; CONGENITAL MYASTHENIC SYNDROME; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME LOCALIZATION; EXCITOTOXICITY; GENE MUTATION; HYPOTHESIS; MORPHOLOGY; MOUSE; MUSCLE CELL; MUSCULAR DYSTROPHY; NERVE FIBER TRANSECTION; NEUROMUSCULAR SYNAPSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; SYNAPSE;

ANIMALS; APOPTOSIS; CALCIUM; DISEASE MODELS, ANIMAL; HUMANS; MICE; MICE, TRANSGENIC; MUSCLE FIBERS; MUSCLE FIBERS, SKELETAL; MUSCLE, SKELETAL; MYASTHENIC SYNDROMES, CONGENITAL; RECEPTORS, CHOLINERGIC;

EID: 33746218974     PISSN: 09699961     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.nbd.2006.04.018     Document Type: Article

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