Both spike and background genes contribute to murine coronavirus neurovirulence

Journal of Virology

Volumn 80, Issue 14, 2006, Pages 6834-6843

  Iacono, Kathryn T ^a   Kazi, Lubna ^a   Weiss, Susan R ^a  

^a UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CELL RECEPTOR; VIRUS SPIKE PROTEIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; CORONAVIRUS; FATALITY; GENE EXPRESSION; HOST CELL; IMMUNE RESPONSE; IMMUNOPATHOGENESIS; MACROPHAGE; MALE; MOUSE; MURINE HEPATITIS CORONAVIRUS; NEUTROPHIL CHEMOTAXIS; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; T LYMPHOCYTE; VIRUS CELL INTERACTION; VIRUS ENCEPHALITIS; VIRUS GENE; VIRUS GENOME; VIRUS HEPATITIS; VIRUS PATHOGENESIS; VIRUS RECOMBINANT; VIRUS SPIKE; VIRUS STRAIN; VIRUS VIRULENCE;

ANIMALS; BRAIN; CORONAVIRUS INFECTIONS; ENCEPHALITIS, VIRAL; GENE EXPRESSION REGULATION, VIRAL; GENES, VIRAL; HEPATITIS, VIRAL, ANIMAL; IMMUNITY, NATURAL; MACROPHAGES; MALE; MICE; MURINE HEPATITIS VIRUS; NEUTROPHIL INFILTRATION; NEUTROPHILS; RECEPTORS, VIRUS; RECOMBINATION, GENETIC; SPECIES SPECIFICITY; T-LYMPHOCYTES; VIRAL PROTEINS;

MURINE HEPATITIS VIRUS;

EID: 33745796824     PISSN: 0022538X     EISSN: None     Source Type: Journal    
DOI: 10.1128/JVI.00432-06     Document Type: Article

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