-
1
-
-
0035044085
-
Neovascularization of ischemic myocardium by human bone-marrow-derived angioblasts prevents cardiomyocyte apoptosis, reduces remodeling and improves cardiac function
-
Kocher A.A., Schuster M.D., Szabolcs M.J., et al. Neovascularization of ischemic myocardium by human bone-marrow-derived angioblasts prevents cardiomyocyte apoptosis, reduces remodeling and improves cardiac function. Nat Med 7 (2001) 430-436
-
(2001)
Nat Med
, vol.7
, pp. 430-436
-
-
Kocher, A.A.1
Schuster, M.D.2
Szabolcs, M.J.3
-
2
-
-
0041358457
-
+ stem cells play a key role in therapeutic angiogenesis induced by bone marrow cell implantation
-
+ stem cells play a key role in therapeutic angiogenesis induced by bone marrow cell implantation. Am J Physiol Heart Circ Physiol 285 (2003) H931-H937
-
(2003)
Am J Physiol Heart Circ Physiol
, vol.285
-
-
Li, T.S.1
Hamano, K.2
Nishida, M.3
-
3
-
-
0037336698
-
Improvement of cardiac function by bone marrow cell implantation in a rat hypoperfusion heart model
-
Nishida M., Li T.S., Hirata K., Yano M., Matsuzaki M., and Hamano K. Improvement of cardiac function by bone marrow cell implantation in a rat hypoperfusion heart model. Ann Thorac Surg 75 (2003) 768-773
-
(2003)
Ann Thorac Surg
, vol.75
, pp. 768-773
-
-
Nishida, M.1
Li, T.S.2
Hirata, K.3
Yano, M.4
Matsuzaki, M.5
Hamano, K.6
-
4
-
-
0034841919
-
Local implantation of autologous bone marrow cells for therapeutic angiogenesis in patients with ischemic heart disease. clinical trial and preliminary results
-
Hamano K., Nishida M., Hirata K., et al. Local implantation of autologous bone marrow cells for therapeutic angiogenesis in patients with ischemic heart disease. clinical trial and preliminary results. Jpn Circ J 65 (2001) 845-847
-
(2001)
Jpn Circ J
, vol.65
, pp. 845-847
-
-
Hamano, K.1
Nishida, M.2
Hirata, K.3
-
5
-
-
0037044406
-
Repair of infarcted myocardium by autologous intracoronary mononuclear bone marrow cell transplantation in humans
-
Strauer B.E., Brehm M., Zeus T., et al. Repair of infarcted myocardium by autologous intracoronary mononuclear bone marrow cell transplantation in humans. Circulation 106 (2002) 1913-1918
-
(2002)
Circulation
, vol.106
, pp. 1913-1918
-
-
Strauer, B.E.1
Brehm, M.2
Zeus, T.3
-
6
-
-
0037058848
-
Transplantation of progenitor cells and regeneration enhancement in acute myocardial infarction (TOPCARE-AMI)
-
Assmus B., Schachinger V., Teupe C., et al. Transplantation of progenitor cells and regeneration enhancement in acute myocardial infarction (TOPCARE-AMI). Circulation 106 (2002) 3009-3017
-
(2002)
Circulation
, vol.106
, pp. 3009-3017
-
-
Assmus, B.1
Schachinger, V.2
Teupe, C.3
-
7
-
-
0037564082
-
Catheter-based autologous bone marrow myocardial injection in no-option patients with advanced coronary artery disease. a feasibility study
-
Fuchs S., Satler L.F., Kornowski R., et al. Catheter-based autologous bone marrow myocardial injection in no-option patients with advanced coronary artery disease. a feasibility study. J Am Coll Cardiol 41 (2003) 1721-1724
-
(2003)
J Am Coll Cardiol
, vol.41
, pp. 1721-1724
-
-
Fuchs, S.1
Satler, L.F.2
Kornowski, R.3
-
8
-
-
0038701741
-
Transendocardial, autologous bone marrow cell transplantation for severe, chronic ischemic heart failure
-
Perin E.C., Dohmann H.F., Borojevic R., et al. Transendocardial, autologous bone marrow cell transplantation for severe, chronic ischemic heart failure. Circulation 107 (2003) 2294-2302
-
(2003)
Circulation
, vol.107
, pp. 2294-2302
-
-
Perin, E.C.1
Dohmann, H.F.2
Borojevic, R.3
-
9
-
-
0037417501
-
Autologous bone-marrow stem-cell transplantation for myocardial regeneration
-
Stamm C., Westphal B., Kleine H.D., et al. Autologous bone-marrow stem-cell transplantation for myocardial regeneration. Lancet 361 (2003) 45-46
-
(2003)
Lancet
, vol.361
, pp. 45-46
-
-
Stamm, C.1
Westphal, B.2
Kleine, H.D.3
-
10
-
-
1842507654
-
Profoundly reduced neovascularization capacity of bone marrow mononuclear cells derived from patients with chronic ischemic heart disease
-
Heeschen C., Lehmann R., Honold J., et al. Profoundly reduced neovascularization capacity of bone marrow mononuclear cells derived from patients with chronic ischemic heart disease. Circulation 109 (2004) 1615-1622
-
(2004)
Circulation
, vol.109
, pp. 1615-1622
-
-
Heeschen, C.1
Lehmann, R.2
Honold, J.3
-
11
-
-
0037434561
-
Circulating endothelial progenitor cells, vascular function, and cardiovascular risk
-
Hill J.M., Zalos G., Halcox J.P., et al. Circulating endothelial progenitor cells, vascular function, and cardiovascular risk. N Engl J Med 348 (2003) 593-600
-
(2003)
N Engl J Med
, vol.348
, pp. 593-600
-
-
Hill, J.M.1
Zalos, G.2
Halcox, J.P.3
-
12
-
-
17644433975
-
Number and migratory activity of circulating endothelial progenitor cells inversely correlate with risk factors for coronary artery disease
-
Vasa M., Fichtlscherer S., Aicher A., et al. Number and migratory activity of circulating endothelial progenitor cells inversely correlate with risk factors for coronary artery disease. Circ Res 89 (2001) E1-E7
-
(2001)
Circ Res
, vol.89
-
-
Vasa, M.1
Fichtlscherer, S.2
Aicher, A.3
-
13
-
-
0037180517
-
Human endothelial progenitor cells from type II diabetics exhibit impaired proliferation, adhesion, and incorporation into vascular structures
-
Tepper O.M., Galiano R.D., Capla J.M., et al. Human endothelial progenitor cells from type II diabetics exhibit impaired proliferation, adhesion, and incorporation into vascular structures. Circulation 106 (2002) 2781-2786
-
(2002)
Circulation
, vol.106
, pp. 2781-2786
-
-
Tepper, O.M.1
Galiano, R.D.2
Capla, J.M.3
-
14
-
-
0033826581
-
The HMG-CoA reductase inhibitor simvastatin activates the protein kinase Akt and promotes angiogenesis in normocholesterolemic animals
-
Kureishi Y., Luo Z., Shiojima I., et al. The HMG-CoA reductase inhibitor simvastatin activates the protein kinase Akt and promotes angiogenesis in normocholesterolemic animals. Nat Med 6 (2000) 1004-1010
-
(2000)
Nat Med
, vol.6
, pp. 1004-1010
-
-
Kureishi, Y.1
Luo, Z.2
Shiojima, I.3
-
15
-
-
0035912855
-
Increase in circulating endothelial progenitor cells by statin therapy in patients with stable coronary artery disease
-
Vasa M., Fichtlscherer S., Adler K., et al. Increase in circulating endothelial progenitor cells by statin therapy in patients with stable coronary artery disease. Circulation 103 (2001) 2885-2890
-
(2001)
Circulation
, vol.103
, pp. 2885-2890
-
-
Vasa, M.1
Fichtlscherer, S.2
Adler, K.3
-
16
-
-
0034898468
-
HMG-CoA reductase inhibitor mobilizes bone marrow-derived endothelial progenitor cells
-
Llevadot J., Murasawa S., Kureishi Y., et al. HMG-CoA reductase inhibitor mobilizes bone marrow-derived endothelial progenitor cells. J Clin Invest 108 (2001) 399-405
-
(2001)
J Clin Invest
, vol.108
, pp. 399-405
-
-
Llevadot, J.1
Murasawa, S.2
Kureishi, Y.3
-
17
-
-
0038814315
-
HMG-CoA reductase inhibitors reduce senescence and increase proliferation of endothelial progenitor cells via regulation of cell cycle regulatory genes
-
Assmus B., Urbich C., Aicher A., et al. HMG-CoA reductase inhibitors reduce senescence and increase proliferation of endothelial progenitor cells via regulation of cell cycle regulatory genes. Circ Res 92 (2003) 1049-1055
-
(2003)
Circ Res
, vol.92
, pp. 1049-1055
-
-
Assmus, B.1
Urbich, C.2
Aicher, A.3
-
18
-
-
20844431696
-
Statin-induced improvement of endothelial progenitor cell mobilization, myocardial neovascularization, left ventricular function, and survival after experimental myocardial infarction requires endothelial nitric oxide synthase
-
Landmesser U., Engberding N., Bahlmann F.H., et al. Statin-induced improvement of endothelial progenitor cell mobilization, myocardial neovascularization, left ventricular function, and survival after experimental myocardial infarction requires endothelial nitric oxide synthase. Circulation 110 (2004) 1933-1939
-
(2004)
Circulation
, vol.110
, pp. 1933-1939
-
-
Landmesser, U.1
Engberding, N.2
Bahlmann, F.H.3
-
19
-
-
0242551506
-
Statin use is associated with enhanced collateralization of severely diseased coronary arteries
-
Pourati I., Kimmelstiel C., Rand W., and Karas R.H. Statin use is associated with enhanced collateralization of severely diseased coronary arteries. Am Heart J 146 (2003) 876-881
-
(2003)
Am Heart J
, vol.146
, pp. 876-881
-
-
Pourati, I.1
Kimmelstiel, C.2
Rand, W.3
Karas, R.H.4
-
20
-
-
15444372697
-
Plaque rupture after short periods of fat feeding in the apolipoprotein E-knockout mouse. model characterization and effects of pravastatin treatment
-
Johnson J., Carson K., Williams H., et al. Plaque rupture after short periods of fat feeding in the apolipoprotein E-knockout mouse. model characterization and effects of pravastatin treatment. Circulation 111 (2005) 1422-1430
-
(2005)
Circulation
, vol.111
, pp. 1422-1430
-
-
Johnson, J.1
Carson, K.2
Williams, H.3
-
21
-
-
0032584177
-
Upregulation of endothelial nitric oxide synthase by HMG CoA Reductase inhibitors
-
Laufs U., La Fata V., Plutzky J., and Liao J.K. Upregulation of endothelial nitric oxide synthase by HMG CoA Reductase inhibitors. Circulation 97 (1998) 1129-1135
-
(1998)
Circulation
, vol.97
, pp. 1129-1135
-
-
Laufs, U.1
La Fata, V.2
Plutzky, J.3
Liao, J.K.4
-
22
-
-
0035200038
-
Hyperglycemia reduces coronary collateral blood flow through a nitric oxide-mediated mechanism
-
Kersten J.R., Toller W.G., Tessmer J.P., Pagel P.S., and Warltier D.C. Hyperglycemia reduces coronary collateral blood flow through a nitric oxide-mediated mechanism. Am J Physiol 281 (2001) H2097-H2104
-
(2001)
Am J Physiol
, vol.281
-
-
Kersten, J.R.1
Toller, W.G.2
Tessmer, J.P.3
Pagel, P.S.4
Warltier, D.C.5
-
23
-
-
1642422782
-
Effect of statin treatment on coronary collateral flow in patients with coronary artery disease
-
Zbinden S., Brunner N., Wustmann K., Billinger M., Meier B., and Seiler C. Effect of statin treatment on coronary collateral flow in patients with coronary artery disease. Heart 90 (2004) 448-449
-
(2004)
Heart
, vol.90
, pp. 448-449
-
-
Zbinden, S.1
Brunner, N.2
Wustmann, K.3
Billinger, M.4
Meier, B.5
Seiler, C.6
-
24
-
-
0035957682
-
Inhibition of endothelial cell migration by cerivastatin, an HMG-CoA reductase inhibitor. contribution to its anti-angiogenic effect
-
Vincent L., Chen W., Hong L., et al. Inhibition of endothelial cell migration by cerivastatin, an HMG-CoA reductase inhibitor. contribution to its anti-angiogenic effect. FEBS Lett 495 (2001) 159-166
-
(2001)
FEBS Lett
, vol.495
, pp. 159-166
-
-
Vincent, L.1
Chen, W.2
Hong, L.3
-
25
-
-
0036120428
-
Cerivastatin, an inhibitor of 3-hydroxy-3-methylglutaryl coenzyme A reductase, inhibits endothelial cell proliferation induced by angiogenic factors in vitro and angiogenesis in in vivo models
-
Vincent L., Soria C., Mirshahi F., et al. Cerivastatin, an inhibitor of 3-hydroxy-3-methylglutaryl coenzyme A reductase, inhibits endothelial cell proliferation induced by angiogenic factors in vitro and angiogenesis in in vivo models. Arterioscler Thromb Vasc Biol 22 (2002) 623-629
-
(2002)
Arterioscler Thromb Vasc Biol
, vol.22
, pp. 623-629
-
-
Vincent, L.1
Soria, C.2
Mirshahi, F.3
-
26
-
-
11144238571
-
3-Hydroxy-3-methylglutaryl-coenzyme a reductase inhibitor, fluvastatin, as a novel agent for prophylaxis of renal cancer metastasis
-
Horiguchi A., Sumitomo M., Asakuma J., Asano T., Asano T., and Hayakawa M. 3-Hydroxy-3-methylglutaryl-coenzyme a reductase inhibitor, fluvastatin, as a novel agent for prophylaxis of renal cancer metastasis. Clin Cancer Res 10 (2004) 8648-8655
-
(2004)
Clin Cancer Res
, vol.10
, pp. 8648-8655
-
-
Horiguchi, A.1
Sumitomo, M.2
Asakuma, J.3
Asano, T.4
Asano, T.5
Hayakawa, M.6
-
27
-
-
0037134840
-
Atorvastatin decreases vascular endothelial growth factor in patients with coronary artery disease
-
Alber H.F., Dulak J., Frick M., et al. Atorvastatin decreases vascular endothelial growth factor in patients with coronary artery disease. J Am Coll Cardiol 39 (2002) 1951-1955
-
(2002)
J Am Coll Cardiol
, vol.39
, pp. 1951-1955
-
-
Alber, H.F.1
Dulak, J.2
Frick, M.3
-
29
-
-
10744220087
-
Statins differentially regulate vascular endothelial growth factor synthesis in endothelial and vascular smooth muscle cells
-
Frick M., Dulak J., Cisowski J., et al. Statins differentially regulate vascular endothelial growth factor synthesis in endothelial and vascular smooth muscle cells. Atherosclerosis 170 (2003) 229-236
-
(2003)
Atherosclerosis
, vol.170
, pp. 229-236
-
-
Frick, M.1
Dulak, J.2
Cisowski, J.3
-
30
-
-
33646835149
-
High-dose atorvastatin improves hypercholesterolemic endothelial dysfunction without improving collateral-dependent myocardial perfusion. role of endostatin and vascular endothelial growth factor (abstract)
-
Boodhwani M., Nakai Y., Voisine P., and Sellke F.W. High-dose atorvastatin improves hypercholesterolemic endothelial dysfunction without improving collateral-dependent myocardial perfusion. role of endostatin and vascular endothelial growth factor (abstract). Circulation 112 Suppl 2 (2005) II-376
-
(2005)
Circulation
, vol.112
, Issue.SUPPL. 2
-
-
Boodhwani, M.1
Nakai, Y.2
Voisine, P.3
Sellke, F.W.4
-
31
-
-
0037233913
-
HMG-CoA reductase inhibitors regulate inflammatory transcription factors in human endothelial and vascular smooth muscle cells
-
Dichtl W., Dulak J., Frick M., et al. HMG-CoA reductase inhibitors regulate inflammatory transcription factors in human endothelial and vascular smooth muscle cells. Arterioscler Thromb Vasc Biol 23 (2003) 58-63
-
(2003)
Arterioscler Thromb Vasc Biol
, vol.23
, pp. 58-63
-
-
Dichtl, W.1
Dulak, J.2
Frick, M.3
-
32
-
-
15444368304
-
HMG-CoA reductase inhibition reduces monocyte CC chemokine receptor 2 expression and monocyte chemoattractant protein-1-mediated monocyte recruitment in vivo
-
Han K.H., Ryu J., Hong K.H., et al. HMG-CoA reductase inhibition reduces monocyte CC chemokine receptor 2 expression and monocyte chemoattractant protein-1-mediated monocyte recruitment in vivo. Circulation 111 (2005) 1439-1447
-
(2005)
Circulation
, vol.111
, pp. 1439-1447
-
-
Han, K.H.1
Ryu, J.2
Hong, K.H.3
|