Caspase-3 mediated cleavage of MEKK1 promotes p53 transcriptional activity

Journal of Molecular and Cellular Cardiology

Volumn 40, Issue 5, 2006, Pages 605-618

  Zebrowski, David C ^a   Alcendor, Ralph R ^a   Kirshenbaum, Lorrie A ^b   Sadoshima, Junichi ^a  

^a RUTGERS NEW JERSEY MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF MANITOBA   (Canada)

Author keywords

Apoptosis; Cardiac myocytes; Caspases 3; Cleavage; Ischemia reperfusion; p53

Indexed keywords

CASPASE 3; MITOGEN ACTIVATED PROTEIN KINASE KINASE KINASE 1; PROTEIN P53; STRESS ACTIVATED PROTEIN KINASE; ACTIVATING TRANSCRIPTION FACTOR 2; ATF2 PROTEIN, RAT; CASP3 PROTEIN, RAT; CASPASE; LUCIFERASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CONTROLLED STUDY; GENETIC TRANSCRIPTION; HEART MUSCLE CELL; HEART MUSCLE ISCHEMIA; IN VITRO STUDY; IN VIVO STUDY; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN PHOSPHORYLATION; RAT; REPERFUSION; SIGNAL TRANSDUCTION; ANIMAL; CELL CULTURE; ISCHEMIA; METABOLISM; PATHOLOGY; PHOSPHORYLATION; REPERFUSION INJURY; WISTAR RAT;

ACTIVATING TRANSCRIPTION FACTOR 2; ANIMALS; CASPASE 3; CASPASES; CELLS, CULTURED; ISCHEMIA; LUCIFERASES; MAP KINASE KINASE KINASE 1; PHOSPHORYLATION; RATS; RATS, WISTAR; REPERFUSION INJURY; SIGNAL TRANSDUCTION; TRANSCRIPTION, GENETIC; TUMOR SUPPRESSOR PROTEIN P53;

EID: 33646092970     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2005.11.012     Document Type: Article

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